Postdoctoral positions are available in the laboratory of Prof. Nicholas Baker, in the Department of Genetics at Albert Einstein College of Medicine in New York, USA. (http://fruitfly4.aecom.yu.edu/index.html). The successful applicant will be interested in molecular and genetic approaches to study growth and/or neural differentiation in vivo using the fruitfly Drosophila melanogaster. We are initiating projects to study the regulation of the cell cycle and other aspects of cell physiology during terminal differentiation of neurons, and the pathways or cell death, cell engulfment, and cell orientation during cell competition and organ growth.
Applicants should have a relevant doctoral degree, a strong research background and a passion for science. Prior experience with Drosophila is not essential. Please send CV including list of publications and names of 3 references by email to email@example.com. Albert Einstein College of Medicine is equal opportunity employer and located in a residential suburb of New York city. Salary will be based on the NIH scale.
selected recent papers:
Li, W. and Baker, N.E. (2007) Engulfment is required for cell competition. Cell, 129, 1215-1225.
Baker, N.E. (2007) Pattern formation and the spatial regulation of proliferation. Curr. Opin. Genet Dev, 17, 287-293.
Baker, N.E. and Li, W. (2008) Cell competition and its possible relation to cancer. Cancer Research 68: 5505-5507.
Bhattacharya, A. and Baker, N.E. (2009) The HLH protein Extramacrochaetae is required for R7 and cone cell fates in the Drosophila eye Dev. Biol. 327, 288-300.
Firth, L.C. and Baker (2009) Retinal determination genes as targets and possible effectors of extracellular signals. Dev. Biol. 327, 366-375.
Fullard, J.F.*, Kale, A.*, and Baker, N.E. (2009). Clearance of apoptotic corpses. Apoptosis, 14: 1029-37.
Baker, N.E., Bhattacharya, A., and Firth, L.C. (2009) Regulation of Hh signal transduction as Drosophila eye differentiation progresses. Dev. Biol. 335, 356-366
Li, W*., Kale, A.*, and Baker, N.E. (2009) Oriented cell division as a response to cell death and cell competition. Curr. Biol. 19 1821-1826