A Database of Drosophila Genes & Genomes

 

Aberration Dmel\Df(2R)03072

General Information
SymbolDmel\Df(2R)03072SpeciesD. melanogaster
NameFlyBase IDFBab0000001
Feature typechromosomal_deletionCreated / Updated2006-08-22/2006-08-22
Formalized genetic data L << bk1 << xen << bk2
Sequence coordinates
Deleted segment51A5--51C1
Duplicated segment
Computed Breakpoints include 51A5;51C1
Breakpoints Inherited  
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Cytological Order
Progenitor
Mutagen
Class of aberration (relative to progenitor)
Breakpoints
Causes alleles
Carries alleles
Transposon Insertions
Genetic mapping information
Comments
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All limits from polytene analysis (FBrf0067338)
 
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DDBJ /
EMBL /
GenBank
DNA sequence
Protein sequence
Name
 
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hide Genes Deleted / Disrupted
Complementation Data
Completely deleted / disrupted
Molecular Data
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Complementation Data
Molecular Data
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Complementation Data
Molecular Data
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Complementation Data
Molecular Data
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hide Phenotypic Data
In combination with other aberrations
NOT in combination with other aberrations
The Df(2R)03072 chromosome does not act as a dominant suppressor of telomeric silencing (assayed using the effect of the chromosome on the eye colour phenotype of flies carrying "P{wvar}KR3-2", a stable "brown-red" variant of the P{3\'WP-2,wvar}2Lt insertion).
hide Position Effect Variegation Data
  
hide Stocks ( 2 )
Bloomington
Kyoto
hide Notes on Origin
Discoverer
hide Balancer / Genotype Variants of the Aberration
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      hide Synonyms & Secondary IDs ( 2 )
      Reported As
      Symbol Synonym
      Name Synonym
      Secondary FlyBase IDs
        hide References ( 12 )
        Research paper
        Roignant et al., 2006, Genes Dev. 20(7): 795--806
        The novel SAM domain protein Aveugle is required for Raf activation in the Drosophila EGF receptor signaling pathway. [FBrf0190559]
        Brumby et al., 2004, Genetics 168(1): 227--251
        A genetic screen for dominant modifiers of a cyclin E hypomorphic mutation identifies novel regulators of S-phase entry in Drosophila. [FBrf0180291]
        Mason et al., 2004, Genetics 168(3): 1353--1370
        A deficiency screen for dominant suppressors of telomeric silencing in Drosophila. [FBrf0180277]
        Moehring and Mackay, 2004, Genetics 167(3): 1249--1263
        The quantitative genetic basis of male mating behavior in Drosophila melanogaster. [FBrf0179348]
        Presgraves, 2003, Genetics 163(3): 955--972
        A fine-scale genetic analysis of hybrid incompatibilities in Drosophila. [FBrf0158988]
        Crozatier et al., 1999, Development 126(19): 4385--4394
        Head versus trunk patterning in the Drosophila embryo; collier requirement for formation of the intercalary segment. [FBrf0111350]
        Spradling et al., 1999, Genetics 153(1): 135--177
        The Berkeley Drosophila genome project gene disruption project. Single P-element insertions mutating 25% of vital Drosophila genes. [FBrf0111489]
        Valcarcel et al., 1999, J. Cell Sci. 112(23): 4389--4396
        Sec61(), a subunit of the protein translocation channel, is required during Drosophila development. [FBrf0112063]
        Prout et al., 1997, Genetics 146(1): 275--285
        Autosomal mutations affecting adhesion between wing surfaces in Drosophila melanogaster. [FBrf0093664]
        Personal communication to FlyBase
        Spradling, 1994.3.9, P element data.
        P element data. [FBrf0075318]
        FlyBase analysis
        FlyBase, 2007, En masse symbol-based assigment of Aberration Class with respect to wild type.
        En masse symbol-based assigment of Aberration Class with respect to wild type. [FBrf0191808]
        Computer file
        BDGP Project Members, 1994-1999, Berkeley Drosophila Genome Project.
        Berkeley Drosophila Genome Project. [FBrf0067338]