64C;65C
Rpd3 << bk1 << Srp54k << sfl << bk2 << sgl
homozygous lethal
Df(3L)ZN47 embryos show defects in tracheal invagination.
The Df(3L)ZN47 chromosome does not act as a dominant suppressor of telomeric silencing (assayed using the effect of the chromosome on the eye colour phenotype of flies carrying "P{wvar}KR3-2", a stable "brown-red" variant of the P{3'WP-2,wvar}2Lt insertion).
Does not cause unconditional lethality in hybrid females when heterozygous with D.simulans chromosome.
No second site non-complementing phenotype with zipEbr and zipmhc-c6.1.
Deficient embryos show an uninterpretable mutant midgut phenotype.
Flies heterozygous for Df(3L)ZN47 with TM3 have incomplete L2, and L4 wing veins and anterior and posterior crossveins. This phenotype is reminiscent of the lost dominant Vn (associated with Df(3L)64C12-D1;65D2-E1) and the recessive vein mutants and is unrelated to ri.
The left Df(3L)ZN47 breakpoint lies in CG12027 or CG4597 or in the region between them, and lies in the range 3L:5012058..5123043 (R5) (predicted cytology: 64C4-6).
The right Df(3L)ZN47 breakpoint lies within Prat2 or CG14820 or in the region between them, and lies in the range 3R:6888496..6907374 (R5) (predicted cytology: 65D2).
All limits from polytene analysis (FBrf0075338)