|Name||Compound (1) Double X||FlyBase ID||FBab0000080|
|Computed Breakpoints include|
|Member of large scale dataset(s)|
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|Nature of the Aberration|
|Class of aberration (relative to progenitor)|
|Formalized genetic data|
|Genetic mapping information|
|Comments on Cytology|
C(1)DX, In(1)dl-49 - In(1)sc8., y f - y- sc8 f..
|Gene Deletion & Duplication Data|
|Genes Deleted / Disrupted|
|Completely deleted / disrupted|
|Genes NOT Deleted / Disrupted|
|Genes NOT Duplicated|
|In combination with other aberrations|
Oocytes from All-Compound females produce anaphase I and meiosis II figures 64% of the time, i.e. they have bypassed metaphase I arrest.
|NOT in combination with other aberrations|
A reversed acrocentric heterozygous for In(1)dl-49; it is useful in balancing because it is very stable, which is probably due to little interstitial heterochromatin. y w f detachments very rarely produced. Also produces a low incidence of homozygosis for w. C(1)DX/0 lethal
|Stocks ( 568 )|
|Notes on Origin|
|Balancer / Genotype Variants of the Aberration|
Some versions also marked with w1.
A study of metaphase arrest found that crossovers between homologs attached to the same centromere do not induce metaphase arrest. Hence exchanges induce metaphase arrest only when they physically conjoin two separate kinetochores. The signal that mediates metaphase arrest is not the exchange event per se, but the resulting tension on homologous kinetochores.
Attached X chromosomes homozygous for various w alleles were used to study dosage compensation of w.
|Synonyms & Secondary IDs ( 6 )|
Compound (1) Double X
|Secondary FlyBase IDs|
|References ( 23 )|
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|Recent research papers (0)|
|All research papers listed in FlyBase were published before 2011|