|Also Known As||A94|
|Computed Breakpoints include||1E3;2B12|
|Member of large scale dataset(s)|
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|All updates||Click here to see a list of all updates to this record from FB2010_08 and on.|
|Nature of the Aberration|
|Class of aberration (relative to progenitor)|
|Formalized genetic data||l(1)1Eb << bk1 << l(1)1Fa << fmf << bk2 << l(1)devl|
|Genetic mapping information|
|Comments on Cytology|
|Gene Deletion & Duplication Data|
|Genes Deleted / Disrupted|
|Completely deleted / disrupted|
|Genes NOT Deleted / Disrupted|
|Genes NOT Duplicated|
|In combination with other aberrations|
|NOT in combination with other aberrations|
In mutants, longitudinal connectives are reduced, also disruptions in motoneural connections are observed. Motor neurons stall and fail to set up the correct innervation pattern in many segments.
Heterozygosity for this deletion suppresses the mutant ovarian phenotype of ovoD2.
Hemizygous embryos were examined with polarised light microscopy and antibody staining: muscle pattern incomplete due to muscle absences, detachments and incorrect attachment sites.
Alleles of png are maternal effect lethal when trans-heterozygous with Df(1)A94. Embryos derived from transheterozygotes mothers also have a giant polyploid nucleus.
|Stocks ( 2 )|
|Notes on Origin|
|Balancer / Genotype Variants of the Aberration|
|Synonyms & Secondary IDs ( 5 )|
|Secondary FlyBase IDs|
|References ( 36 )|
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|Recent research papers (0)|
|All research papers listed in FlyBase were published before 2011|