|Computed Breakpoints include||5A8-5A9;5C5|
|Member of large scale dataset(s)|
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|Nature of the Aberration|
|Class of aberration (relative to progenitor)|
|Formalized genetic data||fs(1)A456 << bk1 << pbr << bk2 << rux|
|Genetic mapping information|
|Comments on Cytology|
|Gene Deletion & Duplication Data|
|Genes Deleted / Disrupted|
|Completely deleted / disrupted|
|Genes NOT Deleted / Disrupted|
|Genes NOT Duplicated|
|In combination with other aberrations|
|NOT in combination with other aberrations|
Does not cause unconditional lethality in hybrid females when heterozygous with D.simulans chromosome.
Transmission rate of Dp(1;f)J21A through females to progeny is 28%, Df(1)C149 has no effect on transmission.
Midgut development of mutant embryos is wild type.
Heterozygosity for this deletion has no effect on the mutant ovarian phenotype of ovoD2.
Hemizygous embryos were examined with polarised light microscopy and antibody staining and found to have wild type muscle pattern and contraction.
|Stocks ( 2 )|
|Notes on Origin|
|Balancer / Genotype Variants of the Aberration|
|Synonyms & Secondary IDs ( 4 )|
Df (1) C149
|Secondary FlyBase IDs|
|References ( 40 )|
|Generate a list of|
|List References by type|
|Recent research papers ( 1 )|