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General Information
Symbol
Df(1)C52
Species
D. melanogaster
Name
FlyBase ID
FBab0000359
Feature type
Computed Breakpoints include
Sequence coordinates
Member of large scale dataset(s)
Nature of Aberration
Cytological Order
Progenitor
Mutagen
Class of aberration (relative to wild type)
Causes alleles
Carries alleles
Transposon Insertions
Formalized genetic data

lz << bk1 << btd << bk2 hits flw

Genetic mapping information
Comments
Comments on Cytology

Limits of break 1 from polytene analysis (FBrf0052647) Left limit of break 2 from polytene analysis (FBrf0034402) Right limit of break 2 from non-inclusion of flw (FBrf0038305)

Sequence Crossreferences
DNA sequence
Protein sequence
Gene Deletion and Duplication Data
Genes Deleted / Disrupted
Genes NOT Deleted / Disrupted
Genes Duplicated
Complementation Data
Completely duplicated
Partially duplicated
Molecular Data
Completely duplicated
Partially duplicated
Genes NOT Duplicated
Complementation Data
 
Molecular Data
 
Affected Genes Inferred by Location
    Phenotypic Data
    In combination with other aberrations
    NOT in combination with other aberrations

    The Keilin's organs are absent in mutants. Homozygous mutant clones induced the larval period fail to survive.

    Embryos hemizygous for Df(1)C52 have a stronger lateral pentascolopidial chordotonal organ (lch5) phenotype than btd alleles, developing up no more than two scolopidia per chordotonal organ, and often developing none.

    No second site non-complementing phenotype with zipEbr and zipmhc-c6.1.

    Shows no maternal enhancement of dpphr4.

    Acts as a dosage sensitive maternal modifier of run : causes tergite defects in greater than 50% of run3 heterozygotes.

    Deficient embryos show a variably penetrant mutant midgut phenotype: incomplete constriction formation.

    Heterozygous larvae have a negative photokinetic stimulus indice similar to wild type and transheterozygotes with lphA2 have an extremely high indice, they are blind.

    Heterozygosity for this deletion has no effect on the mutant ovarian phenotype of ovoD2.

    More than six-fold reduction in sex ratio and significant levels of female lethality is observed when in combination with Sxlf1. Female lethality is also reduced when in combination with da1 but restored when in combination with emcML.

    Mutant wing position of fliF and flw uncovered in deficiency heterozygotes.

    Stocks (1)
    Notes on Origin
    Discoverer

    Lefevre.

     
    Balancer / Genotype Variants of the Aberration
     
    Separable Components
     
    Other Comments
     

    flw alleles are lethal over Df(1)v-L15 but viable over Df(1)C52, with a flap-wing phenotype.

    Synonyms and Secondary IDs (4)
    Reported As
    Name Synonyms
    Secondary FlyBase IDs
      References (47)