|Also Known As||N19|
|Computed Breakpoints include||17A1;18A2|
|Member of large scale dataset(s)|
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|Nature of the Aberration|
|Class of aberration (relative to progenitor)|
|Formalized genetic data||wupA << bk1 << fliH << bk2|
|Genetic mapping information|
|Comments on Cytology|
Ref: Craymer and Roy, 1980, D. I. S. 55: 200--204.
First chromosome breakpoint is 20kb distal to the first chromosome breakpoint of T(1;Y)B137.
|Gene Deletion & Duplication Data|
|Genes Deleted / Disrupted|
|Completely deleted / disrupted|
|Genes NOT Deleted / Disrupted|
|Genes NOT Duplicated|
|In combination with other aberrations|
Transmission rate of Dp(1;f)J21A through females to progeny is 28%, Df(1)N19 weakly decreases transmission.
|NOT in combination with other aberrations|
Does not cause unconditional lethality in hybrid females when heterozygous with D.simulans chromosome.
Deficient embryos show an uninterpretable mutant midgut phenotype.
Homozygous embryos are very abnormal compared to wild-type.
Heterozygosity for this deletion has no effect on the mutant ovarian phenotype of ovoD2.
More than six-fold reduction in sex ratio and significant levels of female lethality is observed when in combination with Sxlf1. Female lethality is also reduced when in combination with da1 but restored when in combination with emcML.
'Sloppy' ventral cord.
|Stocks ( 2 )|
|Notes on Origin|
|Balancer / Genotype Variants of the Aberration|
|Synonyms & Secondary IDs ( 5 )|
|Secondary FlyBase IDs|
|References ( 45 )|
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|Recent research papers (0)|
|All research papers listed in FlyBase were published before 2011|