FB2025_01 , released February 20, 2025
Aberration: Dmel\Df(1)N73
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General Information
Symbol
Df(1)N73
Species
D. melanogaster
Name
FlyBase ID
FBab0000600
Feature type
Computed Breakpoints include
Sequence coordinates
Member of large scale dataset(s)
Nature of Aberration
Cytological Order
Progenitor
Mutagen
Class of aberration (relative to wild type)
Causes alleles
Carries alleles
Transposon Insertions
Formalized genetic data

mus105 << bk1 << Act5C << l(1)5CDa << bk2 << vs

Genetic mapping information
Comments
Comments on Cytology

Second breakpoint not reported.

Limits of break 1 from polytene analysis (FBrf0034402) Left limit of break 2 from inclusion of sqh (FBrf0102325) Right limit of break 2 from polytene analysis (FBrf0034402)

Sequence Crossreferences
DNA sequence
Protein sequence
Gene Deletion and Duplication Data
Genes Deleted / Disrupted
Complementation Data
Partially deleted / disrupted
Molecular Data
Completely deleted
Partially deleted
Genes NOT Deleted / Disrupted
Genes Duplicated
Complementation Data
Completely duplicated
Partially duplicated
Molecular Data
Completely duplicated
Partially duplicated
Genes NOT Duplicated
Complementation Data
 
Molecular Data
 
Affected Genes Inferred by Location (0)
    If no genes are listed here, it may be because the affected region is very large. The JBrowse insert above may show an error for the same reason, and other FlyBase tools such as CytoSearch may also fail for large regions. You can contact FlyBase for more help.
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    Phenotypic Data
    In combination with other aberrations

    Transmission rate of Dp(1;f)J21A through females to progeny is 28%, Df(1)N73 has no effect on transmission.

    NOT in combination with other aberrations

    Homozygous embryos have normal gut morphology.

    Does not cause unconditional lethality in hybrid females when heterozygous with D.simulans chromosome.

    No second site non-complementing phenotype with zipEbr and zipmhc-c6.1.

    Shows no maternal enhancement of dpphr4.

    Dominantly causes tergite defects in less than 50% of run3 heterozygotes.

    Midgut development of mutant embryos is wild type.

    Heterozygosity for this deletion has no effect on the mutant ovarian phenotype of ovoD2.

    No significant change in female lethality when in combination with Sxlf1.

    Hemizygous embryos were examined with polarised light microscopy and antibody staining: muscle attachment abnormality defined.

    Stocks (2)
    Notes on Origin
    Discoverer

    Lefevre.

     
    Balancer / Genotype Variants of the Aberration
     
    Separable Components
     
    Other Comments
     
    Synonyms and Secondary IDs (5)
    Reported As
    Name Synonyms
    Secondary FlyBase IDs
      References (43)