|Name||Deficiency (1) Phosphogluconate dehydrogenase to kurz||FlyBase ID||FBab0000605|
|Also Known As||Df(1)Pgd kz|
|Computed Breakpoints include||2D3;2F5|
|Member of large scale dataset(s)|
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|Nature of the Aberration|
|Class of aberration (relative to progenitor)|
|Formalized genetic data||ph-d << bk1 << aperB << l(1)2Ff << bk2 << phl|
|Genetic mapping information|
Proximal breakpoint within the 17kb EcoRI fragment containing Draf-1. Draf-1 is intact (Nishida et al., 1988)
|Comments on Cytology|
All limits from polytene analysis (FBrf0040957)
|Gene Deletion & Duplication Data|
|Genes Deleted / Disrupted|
|Completely deleted / disrupted|
|Genes NOT Deleted / Disrupted|
|Genes NOT Duplicated|
|In combination with other aberrations|
Df(1)Pgd-kz/In(1)FM7 females show a small but significant increase in X chromosome nondisjunction compared to control +/In(1)FM7 females. Dominant suppressor of variegation of the w gene caused by In(1)wm4 and of variegation at Sb caused by T(2;3)SbV. Dominant enhancer of variegation of the bw gene caused by Dp(?;2)bwD.
|NOT in combination with other aberrations|
|Stocks ( 2 )|
|Notes on Origin|
|Balancer / Genotype Variants of the Aberration|
|Synonyms & Secondary IDs ( 6 )|
Deficiency (1) Phosphogluconate dehydrogenase to kurz
|Secondary FlyBase IDs|
|References ( 30 )|
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|Recent research papers (0)|
|All research papers listed in FlyBase were published before 2011|