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General Information
Symbol
Df(1)RK4
Species
D. melanogaster
Name
FlyBase ID
FBab0000676
Feature type
Computed Breakpoints include

12F5-12F6;13A9-13B1

Sequence coordinates
Member of large scale dataset(s)
Nature of Aberration
Cytological Order
Class of aberration (relative to wild type)
Class of aberration (relative to progenitor)
Breakpoints
Causes alleles
Carries alleles
Transposon Insertions
Formalized genetic data

NetB << bk1 << rut << eag << bk2

Genetic mapping information
Comments
Comments on Cytology

All limits from polytene analysis (FBrf0043474)

Sequence Crossreferences
DNA sequence
Protein sequence
Gene Deletion and Duplication Data
Genes Deleted / Disrupted
Complementation Data
Partially deleted / disrupted
Molecular Data
Completely deleted
Partially deleted
Genes NOT Deleted / Disrupted
Genes Duplicated
Complementation Data
Completely duplicated
Partially duplicated
Molecular Data
Completely duplicated
Partially duplicated
Genes NOT Duplicated
Complementation Data
 
Molecular Data
 
Affected Genes Inferred by Location
    Phenotypic Data
    In combination with other aberrations

    Transmission rate of Dp(1;f)J21A through females to progeny is 28%, Df(1)RK4 weakly decreases transmission.

    NOT in combination with other aberrations

    Some mutant embryos show minor cuticular abnormalities, with the denticle belt typically being interrupted or missing from abdominal segment A4. A larger number of mutant embryos have more severe phenotypes, such as a complete lack of denticle belts. Mouth hook formation is not affected. Extensive embryonic lethality is seen in mutant animals.

    Does not cause unconditional lethality in hybrid females when heterozygous with D.simulans chromosome.

    No second site non-complementing phenotype with zipEbr and zipmhc-c6.1.

    Midgut development of mutant embryos is wild type.

    Heterozygosity for this deletion has no effect on the mutant ovarian phenotype of ovoD2.

    Hemizygous embryos were examined with polarised light microscopy and antibody staining: muscle pattern abnormal due principally to variably expressed defect in insertion site positioning.

    Stocks (2)
    Notes on Origin
    Discoverer

    W.R. Engels.

     
    Balancer / Genotype Variants of the Aberration
     
    Separable Components
     
    Other Comments
     
    Synonyms and Secondary IDs (3)
    Reported As
    Name Synonyms
    Secondary FlyBase IDs
      References (24)