FB2025_01 , released February 20, 2025
Aberration: Dmel\Df(1)sd72b
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General Information
Symbol
Df(1)sd72b
Species
D. melanogaster
Name
Deficiency (1) scalloped
FlyBase ID
FBab0000956
Feature type
chromosomal_deletion
Also Known As
Df(1)sd72b, Df(1)sd72b26, Df(1)sd72b26
Computed Breakpoints include

13F1;14B1

Features within 13F1--14B1
Sequence coordinates
Member of large scale dataset(s)
Nature of Aberration
Cytological Order
Progenitor
Mutagen
X ray
(Campbell et al., 1991)
Class of aberration (relative to wild type)
chromosomal_deletion
(FlyBase, 2007)
Class of aberration (relative to progenitor)
chromosomal_deletion
(Cook et al., 1997, Duffy et al., 1996, Harbecke and Lengyel, 1995, Pauli et al., 1995, Rutherford, 1995, Landecker et al., 1994, Inamdar et al., 1993, Chen et al., 1991, Watson et al., 1991, Kulkarni et al., 1988, Mason et al., 1981, Craymer and Roy, 1980)
Breakpoints

13F1;14B1

(Cook et al., 1997, Duffy et al., 1996, Harbecke and Lengyel, 1995, Pauli et al., 1995, Rutherford, 1995, Landecker et al., 1994, Inamdar et al., 1993, Chen et al., 1991, Watson et al., 1991, Kulkarni et al., 1988, Mason et al., 1981, Craymer and Roy, 1980)
Causes alleles
Carries alleles
Transposon Insertions
Formalized genetic data

bas << bk1 << l(1)A17 << l(1)04 << bk2 << Cyp1

Genetic mapping information
Comments
Comments on Cytology

All limits from polytene analysis (FBrf0034402)

Sequence Crossreferences
DDBJ / EMBL / GenBank
DNA sequence
Protein sequence
Gene Deletion and Duplication Data
Genes Deleted / Disrupted
Complementation Data
Completely deleted / disrupted
Chc
(Katzen and Bishop, 1996, Katzen, 1990)
exd
(Katzen and Bishop, 1996, Harbecke and Lengyel, 1995, Rauskolb et al., 1993, Katzen, 1990, Peifer and Wieschaus, 1990, Tearle and Nusslein-Volhard, 1987)
l(1)04
(Rutherford, 1995)
l(1)13Fa
(Katzen and Bishop, 1996, Katzen, 1990)
l(1)13Fc
(Katzen and Bishop, 1996, Katzen, 1990)
l(1)13Fd
(Katzen and Bishop, 1996, Katzen, 1990)
l(1)14
(Rutherford, 1995)
l(1)14Ad
(Katzen and Bishop, 1996, Rauskolb et al., 1993, Katzen, 1990)
l(1)14Ae
(Katzen and Bishop, 1996, Rauskolb et al., 1993, Katzen, 1990)
l(1)14Af
(Katzen and Bishop, 1996, Rauskolb et al., 1993, Katzen, 1990)
l(1)14Ag
(Katzen and Bishop, 1996, Rauskolb et al., 1993, Katzen, 1990)
l(1)14Ah
(Katzen and Bishop, 1996, Rauskolb et al., 1993, Katzen, 1990)
l(1)14Ai
(Katzen and Bishop, 1996, Rauskolb et al., 1993, Katzen, 1990)
l(1)16
(Rutherford, 1995)
l(1)22
(Rutherford, 1995)
l(1)24
(Rutherford, 1995)
l(1)29
(Rutherford, 1995)
l(1)36
(Rutherford, 1995)
l(1)A10
(Rutherford, 1995)
l(1)A13
(Rutherford, 1995)
l(1)A17
(Rutherford, 1995)
Myb
(Katzen et al., 1998, Katzen and Bishop, 1996, Katzen, 1990)
sd
(Katzen and Bishop, 1996, Katzen, 1990)
shi
(Katzen and Bishop, 1996, Rauskolb et al., 1993, Chen et al., 1991, Katzen, 1990)
slrp
(Homyk and Pye, 1989)
sun
(Kidd et al., 2005, Katzen and Bishop, 1996, Katzen, 1990)
tay
(Katzen and Bishop, 1996, Rauskolb et al., 1993, Katzen, 1990)
Partially deleted / disrupted
Molecular Data
Completely deleted
eIF5
(Marygold et al., 2007)
Partially deleted
Genes NOT Deleted / Disrupted
Genes Duplicated
Complementation Data
Completely duplicated
Partially duplicated
Molecular Data
Completely duplicated
Partially duplicated
Genes NOT Duplicated
Complementation Data
 
Molecular Data
 
Affected Genes Inferred by Location (0)
    If no genes are listed here, it may be because the affected region is very large. The JBrowse insert above may show an error for the same reason, and other FlyBase tools such as CytoSearch may also fail for large regions. You can contact FlyBase for more help.
    In these cases, there will be no "Export to Hitlist" button to the left.
    Phenotypic Data
    In combination with other aberrations

    A slight larval head defect is seen in Df(1)sd72b/Dp(1;Y)shi+1 animals. The bases of the mouth hooks are slightly reduced as are the H-piece and dental sclerites, but the cirri appear to be complete, and the lateral process is not truncated.

    (Mahaffey et al., 2001)

    Transmission rate of Dp(1;f)J21A through females to progeny is 28%, Df(1)sd72b weakly decreases transmission.

    (Cook et al., 1997)
    NOT in combination with other aberrations

    Shows dominant enhancement of dominant haltere phenotype caused by Ubx195 and Ubx9.22.

    (Burnette et al., 1999)

    Does not cause unconditional lethality in hybrid females when heterozygous with D.simulans chromosome.

    (Coyne et al., 1998)

    No second site non-complementing phenotype with zipEbr and zipmhc-c6.1.

    (Halsell and Kiehart, 1998)

    Shows no maternal enhancement of dpphr4.

    (Nicholls and Gelbart, 1998)

    Dominantly causes tergite defects in less than 50% of run3 heterozygotes.

    (Duffy et al., 1996)

    Midgut development of mutant embryos is wild type.

    (Bilder and Scott, 1995)

    Heterozygosity for this deletion has no effect on the mutant ovarian phenotype of ovoD2.

    (Pauli et al., 1995)

    Hemizygous embryos were examined with polarised light microscopy and antibody staining: movement sluggish, muscle pattern incomplete and shape of attachment sites subtly altered.

    (Drysdale et al., 1993)

    Extreme sd phenotype over RpII215Ubl.

    Stocks (2)
    Notes on Origin
    Discoverer

    Lefevre, 26th Feb. 1972.

     

    Separable from: a recessive lethal not closely linked to the deficiency.

    (Fung et al., 2002)
    Balancer / Genotype Variants of the Aberration
     
    Separable Components
     
    Other Comments
     

    Heterozygotes with wild type copy of sd have an incompletely penetrant wing margin phenotype similar to sd. Females often have twisted third thoracic legs. Ectopic bristles are often clustered along the wing veins.

    (Campbell et al., 1991)
    Synonyms and Secondary IDs (10)
    References (54)