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General Information
D. melanogaster
FlyBase ID
Feature type
Computed Breakpoints include


Deleted Segment
Sequence coordinates
Member of large scale dataset(s)
Nature of Aberration
Cytological Order
Class of aberration (relative to wild type)
Class of aberration (relative to progenitor)
Causes alleles
Carries alleles
Transposon Insertions
Formalized genetic data

l(2)38Ea << bk1 << l(2)38Eb << HIS-C << bk2 << Low

Genetic mapping information
Comments on Cytology

Left limit of break 1 from non-inclusion of l(2)38Ea (FBrf0102956) Right limit of break 1 from inclusion of Hr38 (FBrf0102956) Limits of break 2 from polytene analysis (FBrf0039952)

Sequence Crossreferences
DNA sequence
Protein sequence
Gene Deletion and Duplication Data
Phenotypic Data
In combination with other aberrations

The position effect variegation of the w gene seen in In(1)wm4 flies is partially suppressed by one copy of Df(2L)DS6.

NOT in combination with other aberrations

Heterozygous flies show a reduced response of the M pacemaker neurons in the brain to Pdf.

Hemizygous mutants exhibit an acceleration of the S-Phase of mitosis in larval neuroblasts.

The Df(2L)DS6 chromosome does not act as a dominant suppressor of telomeric silencing (assayed using the effect of the chromosome on the eye colour phenotype of flies carrying "P{wvar}KR3-2", a stable "brown-red" variant of the P{3'WP-2,wvar}2Lt insertion).

Dominantly causes tergite defects in less than 50% of run3 heterozygotes.

Heterozygosity for this deletion has no effect on the mutant ovarian phenotype of ovoD2.

Stocks (2)
Notes on Origin
Balancer / Genotype Variants of the Aberration
Separable Components
Other Comments
Synonyms and Secondary IDs (4)
References (33)