FB2025_01 , released February 20, 2025
Aberration: Dmel\Df(2L)E110
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General Information
Symbol
Df(2L)E110
Species
D. melanogaster
Name
FlyBase ID
FBab0001456
Feature type
chromosomal_deletion
Also Known As
E110
Computed Breakpoints include

25F3-26A1;26D3-26D11

Features within 25F3--26D11
Sequence coordinates
Member of large scale dataset(s)
Nature of Aberration
Cytological Order
Progenitor
Mutagen
P-element activity
PM hybrid dysgenesis
(Locke et al., 1988)
Class of aberration (relative to wild type)
chromosomal_deletion
(FlyBase, 2007)
Class of aberration (relative to progenitor)
chromosomal_deletion
(Locke et al., 1988)
Breakpoints

25F3-26A1;26D3-26D11

(Locke et al., 1988)
Causes alleles
Carries alleles
Transposon Insertions
Formalized genetic data

bk1 << E(var)26A << bk2

Genetic mapping information
Comments
Comments on Cytology

All limits from polytene analysis (FBrf0048198)

Sequence Crossreferences
DDBJ / EMBL / GenBank
DNA sequence
Protein sequence
Gene Deletion and Duplication Data
Genes Deleted / Disrupted
Genes NOT Deleted / Disrupted
Genes Duplicated
Complementation Data
Completely duplicated
Partially duplicated
Molecular Data
Completely duplicated
Partially duplicated
Genes NOT Duplicated
Complementation Data
 
Molecular Data
 
Affected Genes Inferred by Location (0)
    If no genes are listed here, it may be because the affected region is very large. The JBrowse insert above may show an error for the same reason, and other FlyBase tools such as CytoSearch may also fail for large regions. You can contact FlyBase for more help.
    In these cases, there will be no "Export to Hitlist" button to the left.
    Phenotypic Data
    In combination with other aberrations

    Dominantly enhances position effect variegation at the w locus caused by In(1)wm4, C(1;YL)wmMc or T(1;4)wm5 and position effect variegation at the y locus caused by In(1)y3P. Position effect variegation at the w locus is not seen when Df(2L)E110 is combined with In(1)wm4rv5, In(1)wm4rv6 or In(1)wm4rv26.

    (Locke et al., 1988)
    NOT in combination with other aberrations

    The Df(2L)E110 chromosome does not act as a dominant suppressor of telomeric silencing (assayed using the effect of the chromosome on the eye colour phenotype of flies carrying "P{wvar}KR3-2", a stable "brown-red" variant of the P{3'WP-2,wvar}2Lt insertion).

    (Mason et al., 2004)

    Heterozygosity for Df(2L)E110 results in 2.6% X chromosome nondisjunction and 0.5% fourth chromosome nondisjunction in In(1)FM7/X ; svspa-pol females.

    (Harris et al., 2003)

    Wild-type clones made in the ovaries of a mutant female, are significantly larger than clones of heterozygous mutant cells. This size difference is greater in clones initiated at 48 hours after egg laying (AEL) than 2 hours AEL.

    (Extavour and Garcia-Bellido, 2001)

    Enhance mottling in wm4.

    (Tartof et al., 1989)

    Homozygous lethal.

    (Locke et al., 1988)

    Enhances position-effect variegation of In(1)wm4. Recessive lethal.

    Stocks (2)
    Notes on Origin
    Discoverer
     
    Balancer / Genotype Variants of the Aberration
     
    Separable Components
     
    Other Comments
     
    Synonyms and Secondary IDs (2)
    References (28)