37D2;38A1
37D2-37E1;37F5-38A1
37D2-37E1;35F5-38A1
37C6-37D1;37F5-38A1
γTub37C << bk1 << l(2)37Da << fs(2)lto3 << bk2 << Lar
Breakpoint(s) molecularly mapped
Fails to complement Df(2L)Exel9043.
No effect on In(1)wm4h position-effect variegation.
Lethal over Df(2L)TW9 and Df(2L)TW158.
Df(2L)E55/Df(2L)TW330 lethal Df(2L)E55/Df(2L)VA15 lethal Df(2L)E55/Df(2L)VA20 lethal Df(2L)E55/Df(2L)VA22 lethal Df(2L)E55/Df(2L)VA23 lethal Df(2L)E55/Df(2L)VA25 lethal Df(2L)E55/Df(2L)TW9 lethal Df(2L)E55/Df(2L)TW12 lethal Df(2L)E55/Df(2L)TW150 viable Df(2L)E55/Df(2L)TW84 viable Df(2L)E55/Df(2L)OD16 viable Df(2L)E55/Df(2L)TW150 non-transparent wing phenotype Df(2L)E55/Df(2L)TW84 non-transparent wing phenotype Df(2L)E55/Df(2L)OD16 non-transparent wing phenotype
Shows no maternal enhancement of dpphr4.
The central nervous system (CNS) is narrower than normal and the commissures are thicker than normal and incompletely separated in Df(2L)E55 embryos.
Dominantly causes tergite defects in less than 50% of run3 heterozygotes.
Midgut development of mutant embryos is wild type.
Homozygous embryos do not always complete head involution.
Heterozygosity for this deletion suppresses the mutant ovarian phenotype of ovoD2.
The Df(2L)E55 chromosome acts as a dominant moderate suppressor of telomeric silencing (assayed using the effect of the chromosome on the eye colour phenotype of flies carrying "P{wvar}KR3-2", a stable "brown-red" variant of the P{3'WP-2,wvar}2Lt insertion), but this is a false positive result (the suppressor is not within the bounds of the deficient region) because the region of the deficiency is covered by one or more nonsuppressing deficiencies.
Ref: FBrf0028752.
Left limit of break 1 from polytene analysis (FBrf0028752) Right limit of break 1 from inclusion of l(2)37Da (citation unavailable) Left limit of break 2 from inclusion of Lar (FBrf0086508) Right limit of break 2 from polytene analysis (FBrf0028752)