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General Information
D. melanogaster
Deficiency (2L) black
FlyBase ID
Feature type
Computed Breakpoints include
Deleted Segment
Sequence coordinates
Member of large scale dataset(s)
Nature of Aberration
Cytological Order
Class of aberration (relative to wild type)
Causes alleles
Carries alleles
Transposon Insertions
Formalized genetic data
bk1 << kuz << vas << bk2 << stc
Genetic mapping information
This deletion removes a single ribosomal protein-coding gene (though other genes are also removed).
Comments on Cytology
Ref: Lindsley and Zimm, 1992
Limits of break 1 from polytene analysis (FBrf0053558) Left limit of break 2 from inclusion of vas (FBrf0077941) Right limit of break 2 from polytene analysis (FBrf0080145)
Sequence Crossreferences
DNA sequence
Protein sequence
Gene Deletion and Duplication Data
Genes Deleted / Disrupted
Genes NOT Deleted / Disrupted
Genes Duplicated
Complementation Data
Completely duplicated
Partially duplicated
Molecular Data
Completely duplicated
Partially duplicated
Genes NOT Duplicated
Complementation Data
Molecular Data
Phenotypic Data
In combination with other aberrations
Inferred to overlap with: Df(2L)BSC345.
NOT in combination with other aberrations
Flies heterozygous for the deletion do not show a Minute bristle phenotype.
Lethal in combination with: Df(2L)BSC147. Inferred to overlap with: Df(2L)BSC147.
Df(2L)b87e25 in combination with a pair of introgressions from D.simulans spanning 30F1-31E7 to 35D7-36A14 Dsim\Int(2L)S and 21A1 to 22D1--23A2 Dsim\Int(2L)D produces semi-sterile male flies. These flies are female sterile.
Suppresses the ommatidial polarity phenotype of dshhs.sev.B. Suppresses the ommatidial polarity phenotype of msnEP549 when driven by Scer\GAL4hs.2sev.
No second site non-complementing phenotype with zipEbr and zipmhc-c6.1.
Shows no maternal enhancement of dpphr4.
Deficient embryos show an uninterpretable mutant midgut phenotype.
Homozygous embryos are very abnormal compared to wild-type.
Stocks (3)
Notes on Origin
The stock contains a second-site deletion uncovering l(2)gl.
Balancer / Genotype Variants of the Aberration
Separable Components
Other Comments
The Df(2L)b87e25 chromosome acts as a dominant weak suppressor of telomeric silencing (assayed using the effect of the chromosome on the eye colour phenotype of flies carrying "P{wvar}KR3-2", a stable "brown-red" variant of the P{3'WP-2,wvar}2Lt insertion), but this is a false positive result (the suppressor is not within the bounds of the deficient region) because the suppressor maps to the tip of 2L rather than to the site of the deficiency. In addition, the deficiency chromosome fails to complement lethal mutations of l(2)gl.
"irradiation" was stated as tentative. "spontaneous" was stated as tentative.
Synonyms and Secondary IDs (11)
References (41)