|Name||Deficiency (2L) clot||FlyBase ID||FBab0001762|
|Also Known As||Df(2L)clot-7, Df(2L)clot7, Df(2L)cl-7, Df(2L)cl7|
|Computed Breakpoints include||25D7;26A7|
|Member of large scale dataset(s)|
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|Nature of the Aberration|
|Class of aberration (relative to progenitor)|
|Formalized genetic data||cype << bk1 << nompC << l(2)26Aa << bk2 << eIF-4a|
|Genetic mapping information|
Breakpoint(s) molecularly mapped
|Comments on Cytology|
All limits from polytene analysis (FBrf0034393)
|Gene Deletion & Duplication Data|
|Genes Deleted / Disrupted|
|Completely deleted / disrupted|
|Genes NOT Deleted / Disrupted|
|Genes NOT Duplicated|
|In combination with other aberrations|
Effect on variegation of y in In(1)y3P and Sb in T(2;3)SbV studied: inactivation of y occurs independently of inactivation of Sb.
Enhances position effect variegation at the w locus caused by In(1)wm4, C(1;YL)wmMc or T(1;4)wm5 and position effect variegation at the y locus caused by In(1)y3P.
Lethal in combination with Df(2L)Gpdh78 (some survivors are seen). Lethal in combination with Df(2L)Gpdh75. Lethal in combination with Df(2L)cl1. Lethal in combination with Df(2L)cl2. Lethal in combination with Df(2L)GpdhA. Viable in combination with T(Y;2)H151. Viable in combination with T(Y;2)D222.
|NOT in combination with other aberrations|
Heterozygosity for this deletion has no effect on the mutant ovarian phenotype of ovoD2.
Enhance mottling in wm4.
|Stocks ( 3 )|
|Notes on Origin|
|Balancer / Genotype Variants of the Aberration|
The presence of a variegating chromosome and a modifier chromosome in the same parental genome can alter the amount the amount of variegation formed in the progeny. The genomic imprinting is not determined by the parental origin of the variegating chromosome but is instead determined by the genetic background the variegating chromosome is subjected to during gametogenesis.
|Synonyms & Secondary IDs ( 8 )|
Deficiency (2L) clot
|Secondary FlyBase IDs|
|References ( 40 )|
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|Recent research papers ( 2 )|