25D2-25D5;25D6-25E1

(Reuter and Szidonya, 1983)

25D2-25D4;25D6-25E1

(George and Terracol, 1997, Pauli et al., 1995, Szidonya and Reuter, 1988)

Homozygous embryos exhibit no dorsal closure and head involution resulting in in the absence of dorsal epidermis. Homozygous embryos and transheterozygous embryos with Df(2L)tkv2 carrying tkv^Ubi-p63E.PB die and possess a vri phenotype. vri³/Df(2L)tkv2 transheterozygotes exhibit shortening of wing vein L2 and L5. Weak dominant enhancer of ea^161.13 embryonic lethal phenotype.

(George and Terracol, 1997)

P{tkv^Brk25D1} has suboptimal levels of expression sufficient to rescue the lethality of tkv⁷/Df(2L)tkv2 through embryonic and larval development. Rescued adults display defects in the wing (cleft in the notum and reduction/deletion of the scutellum) and leg (duplication of ventral structures and loss of dorsal and distal structures) patterning.

(Morimura et al., 1996)

Heterozygosity for this deletion suppresses the mutant ovarian phenotype of ovo^D2.

(Pauli et al., 1995)

Homozygous embryonic lethal, embryos display dorsally open cuticle, severe defective head and contracted epidermis giving the cuticle a rounded appearance. Female transheterozygotes with tkv¹ and tkv⁶ are viable and display a wing phenotype, wing veins are very thick and short along the proximal distal axis. Adult Df(2L)tkv3/tkv⁶ females lay eggs, some of the embryos from these females crossed to wild type males die showing a weakly ventralised phenotype.

(Terracol and Lengyel, 1994)

Homozygous lethal. Chromosome does not have a suppressor effect on w variegation of In(1)w^m4h.

(Reuter and Szidonya, 1983)