A Database of Drosophila Genes & Genomes

FB2013_03, released May 7th, 2013
 

Aberration Dmel\Df(2R)017

General Information
SymbolDmel\Df(2R)017SpeciesD. melanogaster
NameFlyBase IDFBab0001922
Feature typechromosomal_deletion
Also Known AsM(2)017
Computed Breakpoints include 56F5;56F15
Deleted segment56F5--56F15
Sequence coordinates
Member of large scale dataset(s)
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Description
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FB2013_03
FB2013_02
All updates Click here to see a list of all updates to this record from FB2010_08 and on.
hide Nature of the Aberration
Cytological Order
Progenitor
Mutagen
Class of aberration (relative to progenitor)
Breakpoints
Causes alleles
Carries alleles
Transposon Insertions
Formalized genetic data H217 << bk1 << l(2)56Fg << l(2)56Fd << bk2
Genetic mapping information
Comments
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Cytologically invisible deletion.
Small deletion of about 5 bands in the 56F5-15 region.
All limits from polytene analysis (FBrf0032238)
 
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DDBJ /
EMBL /
GenBank
DNA sequence
Protein sequence
Name
 
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Complementation Data
Completely deleted / disrupted
Molecular Data
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Complementation Data
Molecular Data
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Complementation Data
Molecular Data
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Complementation Data
Molecular Data
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In combination with other aberrations
NOT in combination with other aberrations
The Df(2R)017 chromosome does not act as a dominant suppressor of telomeric silencing (assayed using the effect of the chromosome on the eye colour phenotype of flies carrying "P{wvar}KR3-2", a stable "brown-red" variant of the P{3'WP-2,wvar}2Lt insertion).
Dominant short bristle phenotype.
hide Stocks ( 3 )
Bloomington
Kyoto
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Discoverer
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hide Synonyms & Secondary IDs ( 2 )
Reported As
Symbol Synonym
Name Synonym
Secondary FlyBase IDs
hide References ( 23 )
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hide Recent research papers ( 1 )
Nakamoto et al., 2012, Immunity 36(4): 658--667
Virus recognition by toll-7 activates antiviral autophagy in Drosophila. [FBrf0218107]