55A1;55C1-55C3
54F6-55A2;55C1-55C3
54F6-55A1;55C1-55C3
55A1;55C1-55C3
grh << bk1 << l(2)PC4-B << E(dpp)55A << bk2 << hal
Fails to complement Df(2R)PC4, Df(2R)PclXM82 and Df(2R)Pcl7B, complements Df(2R)PC29, Df(2R)P34 and Df(2R)PC66.
The Df(2R)Pcl11B chromosome does not act as a dominant suppressor of telomeric silencing (assayed using the effect of the chromosome on the eye colour phenotype of flies carrying "P{wvar}KR3-2", a stable "brown-red" variant of the P{3'WP-2,wvar}2Lt insertion).
Heterozygosity for Df(2R)Pcl11B results in 0.8% X chromosome nondisjunction and 0.8% fourth chromosome nondisjunction in In(1)FM7/X ; svspa-pol females.
Does not cause unconditional lethality in hybrid females when heterozygous with D.simulans chromosome.
No second site non-complementing phenotype with zipEbr and zipmhc-c6.1.
Uncovers Dsim\jba in D.melanogaster/D.simulans hybrids. Does not uncover Dsim\pm in D.melanogaster/D.simulans hybrids.
Dominantly causes tergite defects in less than 50% of run3 heterozygotes.
Deficient embryos show an uninterpretable mutant midgut phenotype.
Homozygous embryos have a variable number of segments, holes in the epidermis, head defects and disconnected tracheae. The midgut epithelium does not completely surround the yolk, midgut constrictions do not form and Malpighian tubules are not completely elongated and are variable in diameter along their length.
Heterozygosity for this deletion enhances the mutant ovarian phenotype of ovoD2.
Fewer larger nuclei.
When heterozygous with wild type, flies carrying the Pcl deficiency show transformation of second and third legs to first legs. Enhances Pcl3
E.B. Lewis.
Left limit of break 1 from polytene analysis (FBrf0101922) Right limit of break 1 from inclusion of thr (FBrf0065368) Limits of break 2 from polytene analysis (FBrf0038059)