Homozygotes die as wandering third instar larvae.
The
Df(2R)Pcl7B chromosome does not act as a dominant suppressor of telomeric silencing (assayed using the effect of the chromosome on the eye colour phenotype of flies carrying "P{w
var}KR3-2", a stable "brown-red" variant of the
P{3'WP-2,wvar}2Lt insertion).
Heterozygosity for
Df(2R)Pcl7B results in 1.4% X chromosome nondisjunction and 1.4% fourth chromosome nondisjunction in
In(1)FM7/X ;
svspa-pol females.
Shows maternal enhancement of
dpphr4,
dpphr56 and
dpphr27, measured as reduction in recovery of doubly heterozygous progeny.
Dominantly causes tergite defects in less than 50% of
run3 heterozygotes.
Heterozygosity for this deletion enhances the mutant ovarian phenotype of
ovoD2.
When heterozygous with wild type, flies carrying the Pcl deficiency show transformation of second and third legs to first legs. Enhances Pcl3
