54E8-54F1;55B9-55C1
54E8-54F1;55B9-55C1
bk1 << grh << E(dpp)55A << bk2 << sbb
Fails to complement: Df(2R)14H10W-35.
Fails to complement Df(2R)PC4, Df(2R)PclXM82 and Df(2R)Pcl11B, complements Df(2R)PC29, Df(2R)P34 and Df(2R)PC66.
Homozygotes die as wandering third instar larvae.
The Df(2R)Pcl7B chromosome does not act as a dominant suppressor of telomeric silencing (assayed using the effect of the chromosome on the eye colour phenotype of flies carrying "P{wvar}KR3-2", a stable "brown-red" variant of the P{3'WP-2,wvar}2Lt insertion).
Heterozygosity for Df(2R)Pcl7B results in 1.4% X chromosome nondisjunction and 1.4% fourth chromosome nondisjunction in In(1)FM7/X ; svspa-pol females.
No second site non-complementing phenotype with zipEbr and zipmhc-c6.1.
Uncovers Dsim\jba in D.melanogaster/D.simulans hybrids. Does not uncover Dsim\pm in D.melanogaster/D.simulans hybrids.
Dominantly causes tergite defects in less than 50% of run3 heterozygotes.
Heterozygosity for this deletion enhances the mutant ovarian phenotype of ovoD2.
When heterozygous with wild type, flies carrying the Pcl deficiency show transformation of second and third legs to first legs. Enhances Pcl3
E. B. Lewis.
All limits from polytene analysis (FBrf0038059)