60B;60D1

60B8-60B10;60D1-60D2

Lethal in combination with Df(2R)Px5.

Df(2R)Px1/Df(2R)Px2 lethal Df(2R)Px1/Df(2R)Px5 lethal

The Df(2R)Px1 chromosome does not act as a dominant suppressor of telomeric silencing (assayed using the effect of the chromosome on the eye colour phenotype of flies carrying "P{w^var}KR3-2", a stable "brown-red" variant of the P{3'WP-2,w^var}2Lt insertion).

The tracheal phenotypes previously reported for this deficiency in FBrf0051997 and FBrf0072461, where the primary branches are disrupted and discontinuous, are due to the deletion of a gene other than bs, or more than one gene in the region.

Heterozygosity for this deletion has no effect on the mutant ovarian phenotype of ovo^D2.

Wing veins of heterozygote with px-like or Dl-like thickenings, most often near posterior crossvein and free fragments of vein, most often in third posterior cell; L4 vein bent near margin. Wings smaller and narrower than wild type and dusky textured. Closely resembles bs. Expression more extreme in female; enhanced by cold (19^o). homozygous lethal as embryo (Li, 1927).

Complementation tests reported for Df(2R)Px1 may have used a Df(2R)Px2 stock. The Mid-America Stock Center stock 1779 and the Bloomington Stock Center stock 750, which were labeled Df(2R)Px1, were in fact Df(2R)Px2 stocks. The mix-up predates 1991.

FlyBase curator comment: See personal communication from Bloomington Drosophila Stock Center, 2003.8.1, for note about possible confusion between Df(2R)Px1 and Df(2R)Px2 stocks.