42B3-42B4;43E18
43B3-43B5;43E18
42B3-42B4;43E18
43B3-43B5;43E15-43E18
43B3-43B5;43E1-43E8
42B3-42B5;43E15-43E18
43B3;43E18
43B3-43B4;43E18
stmA << bk1 << rambl << bk2
This deletion removes a single ribosomal protein-coding gene (though other genes are also removed).
No effect on In(1)wm4h position-effect variegation.
Flies heterozygous for the deletion do not show a Minute bristle phenotype.
Homozygotes exhibit tracheal defects, with losses of the dorsal drunk, severely disrupted transverse connectives and absences of the visceral branch.
Heterozygosity for this deletion has no effect on the mutant ovarian phenotype of ovoD2.
The ocelli are reduced in size. The number of anterior postalar bristles and ocellar bristles are reduced. The pattern of the interocellar microchaetae is disturbed.
Heterozygotes have small ocelli (Ashburner).
S.I. Tsubota.
Tsubota.
The stock contains a second-site deletion uncovering l(2)gl.
The Df(2R)ST1 chromosome acts as a dominant weak suppressor of telomeric silencing (assayed using the effect of the chromosome on the eye colour phenotype of flies carrying "P{wvar}KR3-2", a stable "brown-red" variant of the P{3'WP-2,wvar}2Lt insertion), but this is a false positive result (the suppressor is not within the bounds of the deficient region) because the region of the deficiency is covered by one or more nonsuppressing deficiencies.
Mistakenly interpreted as carrying a noc allele induced simultaneously.
mistakenly interpreted as carrying a noc allele induced simultaneously
Ref: Bloomington Stock Centre.
Ref: Ashburner et al., 1981, D. I. S. 56: 186--191.
Ref: Lindsley and Zimm, 1987, D. I. S. 65: 1--224
Left limit of break 1 from polytene analysis (FBrf0058613) Right limit of break 1 from polytene analysis (FBrf0085022) Limits of break 2 from polytene analysis (FBrf0036112)