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General Information
Symbol
Df(2R)cn9
Species
D. melanogaster
Name
Deficiency (2R) cinnabar
FlyBase ID
FBab0002170
Feature type
Computed Breakpoints include

42E;44C1

Deleted Segment
Sequence coordinates
Member of large scale dataset(s)
Nature of Aberration
Cytological Order
Progenitor
Class of aberration (relative to wild type)
Causes alleles
Carries alleles
Transposon Insertions
Formalized genetic data

Eb1 << bk1 << so << sax << bk2 << pnut

Genetic mapping information
Comments
Comments on Cytology

Ref: Lindsley and Zimm, 1992

Limits of break 1 from polytene analysis (FBrf0037137) Left limit of break 2 from polytene analysis (FBrf0037137) Right limit of break 2 from non-inclusion of pnut (FBrf0067338)

Sequence Crossreferences
DNA sequence
Protein sequence
Gene Deletion and Duplication Data
Genes Deleted / Disrupted
Complementation Data
Partially deleted / disrupted
Molecular Data
Completely deleted
Partially deleted
Genes NOT Deleted / Disrupted
Genes Duplicated
Complementation Data
Completely duplicated
Partially duplicated
Molecular Data
Completely duplicated
Partially duplicated
Genes NOT Duplicated
Complementation Data
 
Molecular Data
 
Phenotypic Data
In combination with other aberrations
NOT in combination with other aberrations

Heterozygosity for Df(2R)cn9 results in 1.3% X chromosome nondisjunction and 0.9% fourth chromosome nondisjunction in In(1)FM7/X ; svspa-pol females.

No second site non-complementing phenotype with zipEbr and zipmhc-c6.1.

Shows no maternal enhancement of dpphr4.

Dominantly causes tergite defects in less than 50% of run3 heterozygotes.

Heterozygotes with Df(2R)44CE exhibit aberrant migration of the germ cell.

Deficient embryos show an uninterpretable mutant midgut phenotype.

Homozygous embryos show abnormal dorsal closure and tracheae are abnormal.

Heterozygosity for this deletion has no effect on the mutant ovarian phenotype of ovoD2.

Stocks (1)
Notes on Origin
Discoverer
 

The stock contains a second-site deletion uncovering l(2)gl.

Associated with: wunCN.

Recovered as apparent cn double recombinants from test crosses of dysgenic-type males that were heterozygous for a maternally derived dp b cn bw second chromosome and paternally derived autosomes extracted from natural populations and capable of promoting male recombination.

Balancer / Genotype Variants of the Aberration
 
Separable Components
 
Other Comments
 

The Df(2R)cn9 chromosome acts as a dominant weak suppressor of telomeric silencing (assayed using the effect of the chromosome on the eye colour phenotype of flies carrying "P{wvar}KR3-2", a stable "brown-red" variant of the P{3'WP-2,wvar}2Lt insertion), but this is a false positive result (the suppressor is not within the bounds of the deficient region) because the region of the deficiency is covered by one or more nonsuppressing deficiencies.

Synonyms and Secondary IDs (5)
References (45)