|Name||Deficiency (3L) Wrinkled||FlyBase ID||FBab0002357|
|Also Known As||Df(3L)WR10, W10, Df(3L)Wr10|
|Computed Breakpoints include||75A6-75A7;75C2|
|Member of large scale dataset(s)|
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|Nature of the Aberration|
|Class of aberration (relative to progenitor)|
|Formalized genetic data||bk1 << rhea << bk2 << not|
|Genetic mapping information|
Breakpoint(s) molecularly mapped
|Comments on Cytology|
|Gene Deletion & Duplication Data|
|Genes Deleted / Disrupted|
|Completely deleted / disrupted|
|Genes NOT Deleted / Disrupted|
|Genes NOT Duplicated|
|In combination with other aberrations|
|NOT in combination with other aberrations|
Causes inviability or low viability in hybrid females when heterozygous with D.simulans chromosome.
Intermediate second site non-complementing phenotype with zipEbr and zipmhc-c6.1: malformed phenotype penetrance 25-75%.
Deficient embryos show a mutant midgut phenotype: posterior constriction forms anterior to normal site, anterior constriction fails to form.
Homozygous embryos do not complete head involution and germband shortening, and tracheae are disrupted. Maintenance of the midgut epithelium, formation of midgut constrictions and elongation of the Malpighian tubules are variable.
Heterozygosity for this deletion has no effect on the mutant ovarian phenotype of ovoD2.
Number of cells expressing midline glial cell markers in the embryonic CNS is increased by 60%.
Homozygotes show the full cell death defective phenotype.
75B puff is absent.
|Stocks ( 2 )|
|Notes on Origin|
|Balancer / Genotype Variants of the Aberration|
|Synonyms & Secondary IDs ( 13 )|
Deficiency (3L) Wrinkled
|Secondary FlyBase IDs|
|References ( 48 )|
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|Recent research papers (0)|
|All research papers listed in FlyBase were published before 2011|
|Recent reviews (0)|
|All reviews listed in FlyBase were published before 2011|