|Name||Deficiency (3L) vin||FlyBase ID||FBab0002458|
|Also Known As||Df(3L)vin6|
|Computed Breakpoints include||68C8-68C11;69A4-69A5|
|Member of large scale dataset(s)|
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|Nature of the Aberration|
|Class of aberration (relative to progenitor)|
|Formalized genetic data||l(3)68Cc << bk1 << l(3)68Ch << l(3)69Ac << bk2 << l(3)69Ag|
|Genetic mapping information|
|Comments on Cytology|
All limits from polytene analysis (FBrf0080317)
|Gene Deletion & Duplication Data|
|Genes Deleted / Disrupted|
|Completely deleted / disrupted|
|Genes NOT Deleted / Disrupted|
|Genes NOT Duplicated|
|In combination with other aberrations|
|NOT in combination with other aberrations|
The cardial epithelium forms properly in mutant embryos, but falls apart with holes and clustering of cardial cells during its coordinated migration with the dorsal ectoderm. The number of pericardial cells is reduced. Only a small fraction of mutant embryos complete closure of the dorsal ectoderm and they have severe muscle defects.
Homozygous embryos have reduced anal pads. Midgut constrictions do not form, the hindgut is shorter than normal and Malpighian tubules form short buds.
Heterozygosity for this deletion suppresses the mutant ovarian phenotype of ovoD2.
Transheterozygote Df(3L)vin4 Df(3L)vin6 embryos (null for byn) have a major part of the hindgut missing after germ band extension. proctodeum develops aberrantly after germ band extension. Anal pads are also not formed.
Fertile when heterozygous with haync2.
|Stocks ( 4 )|
|Notes on Origin|
|Balancer / Genotype Variants of the Aberration|
|Synonyms & Secondary IDs ( 4 )|
Deficiency (3L) vin
|Secondary FlyBase IDs|
|References ( 24 )|
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|Recent research papers (0)|
|All research papers listed in FlyBase were published before 2011|