68C8;69B4-69B5
68C8-68C12;69B3-69C1
68C8-68C11;69B4-69B5
l(3)68Ca << bk1 << l(3)68Cb << l(3)69Ae << bk2 << gv
This deletion removes a single ribosomal protein-coding gene (though other genes are also removed).
Lethal in combination with Df(3L)BSC120. Inferred to overlap with: Df(3L)BSC120.
Stage 15-16 mutant embryos form a single midgut chamber which is encircled by a thin sheet of visceral muscles. Hindgut development is incomplete.
Flies heterozygous for the deletion do not show a Minute bristle phenotype.
Df(3L)vin7 embryos show defects in tracheal cell migration.
The Df(3L)vin7 chromosome does not act as a dominant suppressor of telomeric silencing (assayed using the effect of the chromosome on the eye colour phenotype of flies carrying "P{wvar}KR3-2", a stable "brown-red" variant of the P{3'WP-2,wvar}2Lt insertion).
Heterozygosity for Df(3L)vin7 results in 7.2% X chromosome nondisjunction and 3.6% fourth chromosome nondisjunction in In(1)FM7/X ; svspa-pol females.
The level of 7-monoenes is normal in heterozygous males. The level of 7,11-dienes is significantly increased in heterozygous females compared to controls.
No second site non-complementing phenotype with zipEbr and zipmhc-c6.1.
Shows no maternal enhancement of dpphr4.
Dominantly causes tergite defects in less than 50% of run3 heterozygotes.
Deficient embryos show a mutant midgut phenotype: no constrictions form, midgut epithelium is thin and fragile.
Homozygous embryos have reduced anal pads. Midgut constrictions do not form, the hindgut is shorter than normal and Malpighian tubules form short buds.
Heterozygosity for this deletion has no effect on the mutant ovarian phenotype of ovoD2.
Fertile when heterozygous with haync2.
Ref: Akam et al., 1978, Cell 13: 215--225
Left limit of break 1 from polytene analysis (FBrf0041709) Right limit of break 1 from inclusion of rt (FBrf0031168) Limits of break 2 from polytene analysis (FBrf0080317)