|Name||Deficiency (3R) Enhancer of split||FlyBase ID||FBab0002571|
|Also Known As||E(spl)RB251, Df(3R)E(spl)RB251, Df(3R)E(spl)R-B251|
|Computed Breakpoints include||96F8-96F9;96F12-96F13|
|Member of large scale dataset(s)|
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|Nature of the Aberration|
|Class of aberration (relative to progenitor)|
|Formalized genetic data||bk1 << l(3)96Fc << l(3)96Fa << bk2|
|Genetic mapping information|
|Comments on Cytology|
|Gene Deletion & Duplication Data|
|Genes Deleted / Disrupted|
|Completely deleted / disrupted|
|Genes NOT Deleted / Disrupted|
|Genes NOT Duplicated|
|In combination with other aberrations|
|NOT in combination with other aberrations|
The number of cells in the nau-expressing muscle precursor clusters is increased compared to wild-type in homozygous embryos and in homozygous embryos derived from homozygous female germline clones (lacking both maternal and zygotic function). The medial clusters expand longitudinally.
Lethal in combination with gro and E(spl)1. Homozygotes have extreme neural hyperplasia and suppress the Nspl-1 phenotype.
|Stocks ( 0 )|
|Notes on Origin|
|Balancer / Genotype Variants of the Aberration|
|Synonyms & Secondary IDs ( 11 )|
Deficiency (3R) Enhancer of split
|Secondary FlyBase IDs|
|References ( 12 )|