|Also Known As||tinΔGC14, tinGC14|
|Computed Breakpoints include||93D6-93D7;93E1|
|Map ( GBrowse )||
No matching regions found.
3R:17,209,495..17,212,798 [+] (Df(3R)GC14:bk1)
|Member of large scale dataset(s)|
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|All updates||Click here to see a list of all updates to this record from FB2010_08 and on.|
|Nature of the Aberration|
|Class of aberration (relative to progenitor)|
|Formalized genetic data||l(3)93Dd << bk1 << Hsrω << tin << bk2 << bap|
|Genetic mapping information|
Breakpoint(s) molecularly mapped
|Comments on Cytology|
Not cytologically detectable.
Chromosome does not puff at 93D upon heat induction.
|Gene Deletion & Duplication Data|
|Genes Deleted / Disrupted|
|Completely deleted / disrupted|
|Genes NOT Deleted / Disrupted|
|Genes NOT Duplicated|
|In combination with other aberrations|
Df(3R)e-Gp4/Df(3R)GC14 lethal Df(3R)e-Gp4/Df(3R)GC14 die at all stages of development
|NOT in combination with other aberrations|
Few somatic gonadal precursors (SGPs) are specified and those that are specified fail to maintain their differentiated state. Germ cells are unable to transfer between germ layers in double mutant embryos with zfh1 (zfh165.34 or zfh175.26). Most germ cells adhere to the endoderm throughout embryogenesis, some scatter near the gut at late stages.
The bridges of fat body that normally span the parasegments at stage 13 fail to form in Df(3R)GC14 embryos, and the fat body tissue remains in a state that morphologically resembles that seen at stage 12 in wild-type embryos.
|Stocks ( 2 )|
|Notes on Origin|
|Balancer / Genotype Variants of the Aberration|
|Synonyms & Secondary IDs ( 8 )|
|Secondary FlyBase IDs|
|References ( 36 )|
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|Recent research papers (0)|
|All research papers listed in FlyBase were published before 2011|
|Recent reviews (0)|
|All reviews listed in FlyBase were published before 2011|