FB2025_01 , released February 20, 2025
Aberration: Dmel\Df(3R)p712
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General Information
Symbol
Df(3R)p712
Species
D. melanogaster
Name
Deficiency (3R) pink
FlyBase ID
FBab0002866
Feature type
deficient_translocation
Computed Breakpoints include

84D4-84D6;85B6;25D

Features within 84D4-6--85B6
Sequence coordinates
Member of large scale dataset(s)
Nature of Aberration
Cytological Order
Progenitor
Mutagen
X ray
Class of aberration (relative to wild type)
deficient_translocation
(FlyBase, 2007)
Class of aberration (relative to progenitor)
deficient_translocation
(Kemphues et al., 1983)
chromosomal_deletion
(Prout et al., 1997, Bhadra and Birchler, 1996, Harbecke and Lengyel, 1995)
Breakpoints

84D4-84D6;85B5;25D

(Kemphues et al., 1983)

84D4-84D6;85B6

(Prout et al., 1997, Bhadra and Birchler, 1996, Harbecke and Lengyel, 1995)
Causes alleles
Carries alleles
Transposon Insertions
Formalized genetic data

l(3)02732 << bk1 << l(3)neo35 << p << bk2 << l(3)00281 << bk3 << stck

Genetic mapping information
Comments
Comments on Cytology

Limits of break 1 from polytene analysis (citation unavailable) Limits of break 2 from polytene analysis (FBrf0082073) Limits of break 3 from polytene analysis (citation unavailable)

Sequence Crossreferences
DDBJ / EMBL / GenBank
DNA sequence
Protein sequence
Gene Deletion and Duplication Data
Genes Deleted / Disrupted
Genes NOT Deleted / Disrupted
Genes Duplicated
Complementation Data
Completely duplicated
Partially duplicated
Molecular Data
Completely duplicated
Partially duplicated
Genes NOT Duplicated
Complementation Data
 
Molecular Data
 
Affected Genes Inferred by Location (0)
    If no genes are listed here, it may be because the affected region is very large. The JBrowse insert above may show an error for the same reason, and other FlyBase tools such as CytoSearch may also fail for large regions. You can contact FlyBase for more help.
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    Phenotypic Data
    In combination with other aberrations
    NOT in combination with other aberrations

    The distal half of the salivary gland turns but the proximal half does not in embryos homozygous for Df(3R)p712 by stage 14.

    (Jattani et al., 2009)

    Has a partially penetrant dominant cuticle phenotype.

    (Cox et al., 2000)

    Dominantly enhances the KrIf-1/+ eye phenotype.

    (Carrera et al., 1998)

    Intermediate second site non-complementing phenotype with zipmhc-c6.1 (though not zipEbr): malformed phenotype penetrance 25-75%.

    (Halsell and Kiehart, 1998)

    Shows no maternal enhancement of dpphr4.

    (Nicholls and Gelbart, 1998)

    Deficient embryos show an uninterpretable mutant midgut phenotype.

    (Bilder and Scott, 1995)

    Homozygous embryos are very abnormal compared to wild-type and inner organs are reduced in size.

    (Harbecke and Lengyel, 1995)
    Stocks (3)
    Notes on Origin
    Discoverer
     
    Balancer / Genotype Variants of the Aberration
     
    Separable Components
     
    Other Comments
     

    The Df(3R)p712 chromosome acts as a dominant weak suppressor of telomeric silencing (assayed using the effect of the chromosome on the eye colour phenotype of flies carrying "P{wvar}KR3-2", a stable "brown-red" variant of the P{3'WP-2,wvar}2Lt insertion), but this is a false positive result (the suppressor is not within the bounds of the deficient region) because the region of the deficiency is covered by one or more nonsuppressing deficiencies.

    (Mason et al., 2004)
    Synonyms and Secondary IDs (4)
    References (39)