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General Information
D. melanogaster
Deficiency (3R) rosy
FlyBase ID
Feature type
Also Known As
Computed Breakpoints include
Sequence coordinates
Member of large scale dataset(s)
Nature of Aberration
Cytological Order
Class of aberration (relative to wild type)
Causes alleles
Carries alleles
Transposon Insertions
Formalized genetic data

Men << bk1 << l(3)87Da << l(3)87Em << bk2 << sqd

Genetic mapping information
Comments on Cytology

Left limit of break 1 from polytene analysis (FBrf0063630) Right limit of break 1 from polytene analysis (FBrf0028767) Left limit of break 2 from polytene analysis (FBrf0063630) Right limit of break 2 from polytene analysis (FBrf0028767)

Sequence Crossreferences
DNA sequence
Protein sequence
Gene Deletion and Duplication Data
Genes Deleted / Disrupted
Genes NOT Deleted / Disrupted
Genes Duplicated
Complementation Data
Completely duplicated
Partially duplicated
Molecular Data
Completely duplicated
Partially duplicated
Genes NOT Duplicated
Complementation Data
Molecular Data
Affected Genes Inferred by Location
    Phenotypic Data
    In combination with other aberrations
    NOT in combination with other aberrations

    When P{P-Sal}Pak3P-Sal is present, the w gene of the P{lacW}Dplac insertion variegates, resulting (in a w- background) in an eye that is mostly white with a few patches of red ommatidia. This P-element-dependent silencing of P{lacW}Dplac is dominantly suppressed by Df(3R)ry27.

    The Df(3R)ry27 chromosome does not act as a dominant suppressor of telomeric silencing (assayed using the effect of the chromosome on the eye colour phenotype of flies carrying "P{wvar}KR3-2", a stable "brown-red" variant of the P{3'WP-2,wvar}2Lt insertion).

    Homozygous embryos show abnormal dorsal closure and tracheal formation.

    Heterozygosity for this deletion has no effect on the mutant ovarian phenotype of ovoD2.

    Homozygous viable. Transheterozygotes with B52ED produce very few adult progeny and those that survive have head defects that resemble those observed by in extreme Dfd mutants. This effect is due to an uncharacterised genetic element on the Df(3R)ry27 chromosome.

    Stocks (2)
    Notes on Origin



    in cu kar chromosome.

    Balancer / Genotype Variants of the Aberration
    Separable Components
    Other Comments
    Synonyms and Secondary IDs (4)
    Reported As
    Name Synonyms
    Deficiency (3R) rosy
    Secondary FlyBase IDs
      References (18)