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General Information
Symbol
Df(4)G
Species
D. melanogaster
Name
Deficiency (4) of Gloor and Green
FlyBase ID
FBab0002954
Feature type
Computed Breakpoints include

102E2-102E7;102E-102F2

Deleted Segment
Sequence coordinates
4:721,173..776,474 (Df(4)G:bk1)
4:1,172,469..1,350,131 (Df(4)G:bk2)
Member of large scale dataset(s)
Nature of Aberration
Cytological Order
Progenitor
Mutagen
Class of aberration (relative to wild type)
Class of aberration (relative to progenitor)
Breakpoints
Causes alleles
Carries alleles
Transposon Insertions
Formalized genetic data
Genetic mapping information
Comments

The 4:1172469..1350131 release 6 coordinates of the right breakpoint are estimates. The left extent corresponds to the left end of pho, which published results say is deleted. The right extent corresponds to the right end of chromosome 4 genome assembly.

The 4:721173..776474 release 6 coordinates of the left breakpoint are estimates. The left extent corresponds to the right end of ey, which published results say is not deleted. The right extent corresponds to the right end of bt, which published results say is deleted.

Comments on Cytology
Sequence Crossreferences
DNA sequence
Protein sequence
Gene Deletion and Duplication Data
Genes Deleted / Disrupted
Genes NOT Deleted / Disrupted
Genes Duplicated
Complementation Data
Completely duplicated
Partially duplicated
Molecular Data
Completely duplicated
Partially duplicated
Genes NOT Duplicated
Complementation Data
 
Molecular Data
 
Phenotypic Data
In combination with other aberrations

Df(4)G/Df(4)3 lethal Df(4)G/Df(4)11 lethal Df(4)G/Df(4)12 lethal Df(4)G/Df(4)24 lethal Df(4)G/Df(4)34 lethal

NOT in combination with other aberrations

Df(4)G does not uncover hybrid sterility when heterozygous in males containing the D.simulans 4th chromosome introgressed into an otherwise D.melanogaster background.

Shows dominant enhancement of dominant haltere phenotype caused by Ubx195 and Ubx9.22.

The rate of transmission of the Df(4)G chromosome is similar to that of a normal fourth chromosome.

Embryos show normal axon guidance.

Heterozygosity for this deletion has no effect on the mutant ovarian phenotype of ovoD2.

Lethal homozygous

Stocks (3)
Notes on Origin
Discoverer

Gloor and Green, 1957.

 
Balancer / Genotype Variants of the Aberration
 
Separable Components
 
Other Comments
 
Synonyms and Secondary IDs (5)
Reported As
Name Synonyms
Deficiency (4) of Gloor and Green
Secondary FlyBase IDs
    References (30)