h42-h43;59E1
h42-h43;59D1-59E2
41A-41B;59D6-59E1
l(2)41Aa << bk1 << msf << bw << bk2 << mi
The In(2R)bwVDe2 pigmentation phenotype is suppressed by C(1;YL)wmMc or In(1)wm51b.
The bw+ cis-effect in different inversion chromosomes is ranked: In(2R)bwVDe1 > In(2R)bwVDe2 > In(2R)bwVK.
Position effect variegation (PEV) at the bw locus caused by In(2R)bwVDe2 is dominantly enhanced by Dp(2;2)Mdh, and suppressed by Df(2R)en-A.
The position effect variegation at the bw locus caused by In(2R)bwVDe2 is suppressed by Df(3R)Ace-HD1.
The position effect variegation of bw caused by In(2R)bwVDe2 is consistently suppressed by T(2;3)SbV, although In(2R)bwVDe2 has little effect on the position effect variegation of Sb caused by T(2;3)SbV. In(2R)bwVDe2 and C(1;YL)wmMc, and In(2R)bwVDe2 and In(1)wm51b in combination show dominant suppression of variegation of one or both of w and bw, as measured by eye pigment levels. In contrast In(2R)bwVDe2 and T(1;4)wmJ in combination show enhancement of variegation of one or both of w and bw.
Homozygous lethal.
Carnitine compounds (L-Carnitine, L-Acetylcarnitine and L-Propionylcarnitine) show a significant suppression on bw variegation. Butyrate gives a weaker suppression.
homozygous lethal in embryonic stage <up>e.g., In(2R)bwDe2 (Tsai, 1955, Genetics 40: 601)</up>. Eye color mosaic of brown and dark brown patches.
Demerec, 14th Oct. 1933.
Choi, 1977, D. I. S. 52: 88; Mettler, Voelker and Mukai, 1977,
Ref: FBrf0066905.
Ref: FBrf0003816 and FBrf0054171.
Limits of break 1 from polytene analysis (FBrf0092594) Left limit of break 2 from complementation mapping against bw (citation unavailable) Right limit of break 2 from polytene analysis (FBrf0098579)