h1-h17
C(1;YL)wmMc/0 ; T(Y;3)A78/+ males are fertile. Suppresses the In(2R)bwVDe2 pigmentation phenotype.
The position effect variegation of Sb caused by T(2;3)SbV is strongly suppressed by C(1;YL)wmMc in both males and females. This interaction is both dominant and dosage sensitive; one copy of the C(1;YL)wmMc chromosome is sufficient to suppress variegation of Sb caused by T(2;3)SbV, however, two copies of C(1;YL)wmMc produce slightly greater suppression than one copy. The T(2;3)SbV chromosome may have a slight suppressing effect on the variegation of w caused by C(1;YL)wmMc in males. In(2R)bwVDe2 and C(1;YL)wmMc in combination show dominant suppression of variegation of one or both of w and bw, as measured by eye pigment levels.
C(1;YL)wmMc/0 males are sterile.
Eyes have a "peppered" phenotype - flecks of dark brown pigmented ommatidia are scattered throughout the wild type surface of the eye.
The chromosome referred to as "In(1)wmMc" in this paper is actually C(1;YL)wmMc (this chromosome is a descendent of In(1)wmMc which carries an X.YL translocation - see FBrf0123212 and Talbert and Henikoff, 2000.1.29, personal communication).
The chromosome referred to as "white<up>mottled McLean</up>" in this paper is actually C(1;YL)wmMc (this chromosome is a descendent of In(1)wmMc which carries an X.YL translocation - see FBrf0123212 and Talbert and Henikoff, 2000.1.29, personal communication).
The chromosome referred to as "wmMc" in this paper is actually C(1;YL)wmMc (this chromosome is a descendent of In(1)wmMc which carries an X.YL translocation - see FBrf0123212 and Talbert and Henikoff, 2000.1.29, personal communication).
X.YL translocation with the point of exchange in the rDNA.