A Database of Drosophila Genes & Genomes

FB2012_01, released January 20th, 2012
 

Aberration Dmel\Df(3L)B71

General Information
SymbolDmel\Df(3L)B71SpeciesD. melanogaster
NameFlyBase IDFBab0029520
Feature typeassortment_derived_deficiency
Computed Breakpoints include
Deleted segment3Lt--61B
Sequence coordinates
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Description
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FB2012_01
FB2011_10
All updates Click here to see a list of all updates to this record from FB2010_08 and on.
hide Nature of the Aberration
Cytological Order
Progenitor
Mutagen
Class of aberration (relative to progenitor)
Breakpoints
Causes alleles
Carries alleles
Transposon Insertions
Formalized genetic data
Genetic mapping information
Comments
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DDBJ /
EMBL /
GenBank
DNA sequence
Protein sequence
Name
 
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Complementation Data
Completely deleted / disrupted
Molecular Data
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Complementation Data
Molecular Data
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Complementation Data
Molecular Data
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Complementation Data
Molecular Data
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In combination with other aberrations
NOT in combination with other aberrations
The Df(3L)B71 chromosome does not act as a dominant suppressor of telomeric silencing (assayed using the effect of the chromosome on the eye colour phenotype of flies carrying "P{wvar}KR3-2", a stable "brown-red" variant of the P{3'WP-2,wvar}2Lt insertion).
hide Stocks ( 2 )
Bloomington
Kyoto
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Discoverer
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Reported As
Symbol Synonym
Ts(YSt;3Rt)B71
 
Name Synonym
Secondary FlyBase IDs
hide References ( 3 )
Research paper
Mason et al., 2004, Genetics 168(3): 1353--1370
A deficiency screen for dominant suppressors of telomeric silencing in Drosophila. [FBrf0180277]
Brill et al., 2000, Development 127(17): 3855--3864
A phospholipid kinase regulates actin organization and intercellular bridge formation during germline cytokinesis. [FBrf0129743]
Personal communication to FlyBase
Brill, 2000.8.14, Deficiency stocks.
Deficiency stocks. [FBrf0131209]