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FB2025_02
,
released April 17, 2025
A set of ~800 largely isogenic deficiency stocks created by FLP-induced recombination between FRT-carrying transgenic insertions; molecularly defined deletion endpoints correspond to initial location of the progenitor insertions. Designed to fill gaps in deletion coverage and breakpoint placement; also used to replace older available deficiencies that have not been molecularly mapped.
Breakpoint based on release 3 sequence coordinate from Thibault et al., 2004, Supplementary Table 3 (FBrf0174228), converted to release 5 coordinate.
Breakpoint from FlyBase's release 5 sequence location of progenitor insertion.
Df(3R)BSC751/Df(3R)E(spl)δ-6 mutant embryos display a significant increase in the number of Ap neurons, as compared with controls.
Dp(3;2)E(spl)-m3null/Df(3R)BSC751, Dp(3;2)E(spl)-m3null,mδnull/Df(3R)BSC751 or Dp(3;2)E(spl)-m3null,mβnull/Df(3R)BSC751 mutant embryos do not display any significant change in the number of Ap neurons, as compared with controls.
Df(3R)BSC751/+ mutant embryos do not display any significant change in the number of Ap neurons, as compared with controls.
The presence of P+PBac{XP5.WH5}BSC751 was verified using the PCR methods and primers described in FBrf0175003.
Exelixis, Inc. determined the insertion site of P{XP}d07975 to be Release 3 genomic coordinate 21870411 on chromosome arm 3R. This corresponds to 96F10 on the Release 3 and Release 5 genome maps. The predicted position of PBac{WH}f01987 on the Release 5 map is 96F8. Consequently, the cytological breakpoints of Df(3R)BSC751 are predicted to be 96F8;96F10.