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General Information
Symbol
Dmel\AntpR
Species
D. melanogaster
Name
of Rapoport
FlyBase ID
FBal0000615
Feature type
allele
Associated gene
Associated Insertion(s)
Carried in Construct
Allele class
Mutagen
Nature of the Allele
Allele class
Mutagen
Mutations Mapped to the Genome
 
Type
Location
Additional Notes
References
Associated Sequence Data
DNA sequence
Protein sequence
 
 
Progenitor genotype
Cytology
Nature of the lesion
Statement
Reference
Caused by aberration
Expression Data
Reporter Expression
Additional Information
Statement
Reference
 
Marker for
Reflects expression of
Reporter construct used in assay
Human Disease Associations
Disease Ontology (DO) Annotations
Models Based on Experimental Evidence ( 0 )
Disease
Evidence
References
Modifiers Based on Experimental Evidence ( 0 )
Disease
Interaction
References
Comments on Models/Modifiers Based on Experimental Evidence ( 0 )
 
Phenotypic Data
Phenotypic Class
Phenotype Manifest In
Detailed Description
Statement
Reference

Transformation of antenna to prothoracic leg identity.

Segments added to antennae, usually distal to aristae. Claw occasionally at end of antenna. Asymmetry pronounced. Rarely an antenna-like organ on sternopleura. Enhanced by D1. Lethal in combination with AntpB (Von Halle, cited in FBrf0066905). RK3A.

External Data
Interactions
Show genetic interaction network for Enhancers & Suppressors
Phenotypic Class
Other
Statement
Reference
Phenotype Manifest In
Additional Comments
Genetic Interactions
Statement
Reference

In heterozygous combination with ctL188 or ctC145, ectopic structures project (usually unilaterally) from the position of the anterior spiracle on the dorsal prothorax, and are associated with malformation of absence of the two macrochaetaes on the humeral callus. The phenotype is incompletely penetrant and not as severe as for Antp11.

Homeotic transformation of antennae to T2 leg seen in AntpR flies can be redirected by pbhs.PB from ectopic leg to maxillary palp.

Xenogenetic Interactions
Statement
Reference
Complementation and Rescue Data
Fails to complement
Comments
Images (0)
Mutant
Wild-type
Stocks (2)
Notes on Origin
Discoverer

Rappaport, 1963.

Comments
Comments

Variable expression but expressivity 100% in combination with D. Moderate dominant allele.

The wing positioning defects in In(3R)AntpR flies were previously attributed to an effect on Antp (FBrf0107412 and FBrf0155711), however, complementation analysis in FBrf0204343 indicates that the Glut4EF gene is also disrupted by the In(3R)AntpR chromosome and suggests that the wing positioning defects are caused instead by an effect on Glut4EF.

FlyBase curator comment: FBrf0155711 attributes the wing positioning defects in In(3R)AntpR flies to an effect on Antp. However, complementation analysis in FBrf0204343 indicates that the Glut4EF gene is also disrupted by the In(3R)AntpR chromosome and suggests that the wing positioning defects are caused instead by an effect on Glut4EF.

FlyBase curator comment: FBrf0107412 attributes the wing positioning defects in In(3R)AntpR flies to an effect on Antp. However, complementation analysis in FBrf0204343 indicates that the Glut4EF gene is also disrupted by the In(3R)AntpR chromosome and suggests that the wing positioning defects are caused instead by an effect on Glut4EF.

External Crossreferences and Linkouts ( 0 )
Synonyms and Secondary IDs (4)
Reported As
Symbol Synonym
AntpR
ssA
Name Synonyms
of Rapoport
spineless-Aristapedia Dominant
Secondary FlyBase IDs
    References (11)