homozygotes do not show significant mortality during the pharate stage.
transheterozygotes exhibit a cut-wing phenotype, as compared to controls
/+ wings are similar to wild type.
mutant late third instar wing discs have disturbed dorsal-ventral (DV) boundaries compared to controls. The anterior-posterior boundary is unaffected.
wings have a notched edge.
mutants show wing margin defects.
mutant animals develop "cut" wings, missing regions of the wing margin.
mutants exhibit a jagged or cut appearance of the fly wing edge.
Hemizygotes show a reduction in the numbers of wing margin bristles and and incised wing margin and wing blade.
Severe cuts in the wing margin.
Shows no interaction with pb
Homozygotes lack wing margin tissue in distal and posterior regions of the wing.
is strongly suppressed by mod(mdg4)ul
: only 20% of flies have one to five gaps in the wing margin. Strong and weak su(Hw)
mutations, with or without mod(mdg4)ul
, completely suppress the ct6
slightly suppress the phenotype of ct6
unless in combination with mod(mdg4)ul
, when the phenotype is completely suppressed.
Scalloped wing margin and lack of many sensory bristles at the anterior wing margin. All non-innervated hairs of the posterior wing margin are absent.
Homozygotes display cut wing and head capsule defects.
wing and vibrissae phenotype.
Geotaxis score significantly lower than for Canton S in a Hirsch-type geotaxis maze.
When in combination with sc alleles ct6
exhibited excisions and hair reduction on the external edge of the wing.
Wings are cut to points, and the wing margins are scalloped.
Scalloped wings. Vibrissae are missing. Completely complemented by Df(1)R19
Cell death observed in prepupal wing bud.
Wing bud narrower than wild type as early as just after eversion of wing in early pupa.
Cut wing character uniform and reliable; usually lacks the abdomen, antenna, arista, and eye effects of ct
. Vibrissae gone or displaced downward to bottom of eye. RK1.