FlyBase Allele Report: Dmel\Dl[9P]

(Pandey et al., 2018, Acar et al., 2008, Christoforou et al., 2008, Marenda et al., 2004, Lai and Rubin, 2001, Sturtevant and Bier, 1995, Vassin et al., 1985)

wing vein | ectopic

(Tsuda et al., 2002)

wing vein L2

(Xie et al., 2012)

Detailed Description

Statement

Reference

Dl^9P heterozygotes show a fully penetrant wing vein thickening phenotype and extra wing vein material phenotypes, as compared to controls.

(Pandey et al., 2018)

Dl^9P/+ flies display thickened wing veins (typically for veins L2 and L5).

(Xie et al., 2012)

Mutant embryos show a complete loss of pericardial cells and hematopoietic precursors, while cardioblast numbers are dramatically increased compared to wild type.

(Grigorian et al., 2011)

Dl^9P mutant neuroectodermal cells transplanted into wild-type hosts do not affect cell fate changes in NB1-1, MP2 and NB7-1 lineages.

RP2/RP2sib and vMP2/dMP2 cell fates are not correctly resolved in Dl^9P mutants.

(Udolph et al., 2009)

The accumulation of hemocytes is normal in the wings of heterozygous pupae, both at 22 and 26 hours after puparium formation.

Heterozygotes show wing vein thickening.

(Christoforou et al., 2008)

20% of heterozygotes have ectopic or duplicated macrochaetae.

(Armstrong et al., 2005)

In Dl^9P homozygous embryos, the early steps of proventricular development including the formation of the ball-like evagination at the ectoderm/endoderm boundary occur normally. However, at stage 14, the anterior boundary cells of the keyhole region fail to invaginate into the endodermal cell layer, but arrest anteriorly and do not move inwards until the final stages of embryonic development (16 and 17). In addition, the posterior boundary cells of the endodermal component of the proventriculus rim collapses in these animals.

(Fuss et al., 2004)

Heterozygotes display extra vein material particularly at the distal tip of veins.

(Lai and Rubin, 2001)

Homozygous mutant somatic clones in the notum give a phenotype of adjacent bristles with no intervening epidermal cells. A band of mutant epidermis, that averages about 4.2 cells wide around the clone, have a wild-type phenotype.

(Renaud and Simpson, 2001)

Dl^9P/Dl^RF animals shifted to the nonpermissive temperature during the early pupal stage develop double shaft bristles.

(Barolo et al., 2000)

Homozygous clones in the scutum produce a similar phenotype to homozygous Dl^RevF10 clones.

(Zeng et al., 1998)

Heterozygotes with Ser^Bd-3 are viable and exhibit a severe wing phenotype.

(Hukriede and Fleming, 1997)

Small effect on the Brd¹ and Brd³ bristle multiplication phenotype.

(Leviten and Posakony, 1996)

Clusters of R8 cells develop in Dl^RF/Dl^9P flies raised at the restrictive temperature. The clusters are not randomly arranged, but are based on the original R8 array.

(Baker and Zitron, 1995)

Instead of forming distinct invagination folds, the Dl mutant stomodeal nervous system anlage invaginates en masse.

(Gonzalez-Gaitan and Jäckle, 1995)

Thickened vein mutant.

(Sturtevant and Bier, 1995)

Hyperplasia of replicating sensory precursors: due to an increased number of ectodermal cells being recruited as sensory precursor cells.

(Bodmer et al., 1993)

In double mutant clones with N^l1N-ts1, both wild type and mutant bristles are formed along the mosaic borders, and occasionally a mutant and a wild type bristle are found adjacent to each other (which never happens in either single mutant). Homozygous N^Ax-59b mutant clones develop as epidermis, and this requires Dl function.

(Heitzler and Simpson, 1993)

Dl^9P/+ flies carrying one or two copies of Egfr^E1 or Egfr^E3 often have blistered wings.

(Baker and Rubin, 1992)

Clones in the thorax have an abnormal distribution of microchaetae and macrochaetae.

(Simpson et al., 1992)

Increase in SMCs per cluster in embryos lacking the maternal product.

(Goriely et al., 1991)

Mutant clones cause the differentiation of masses of adjacent microchaetae not separated by epidermal hairs. On the scutum cells adopt a neural fate. Mutant cells behave nonautonomously: cells produce epidermis only along the mosaic border where they are in contact with wild type cells.

(Heitzler and Simpson, 1991)

Severe "abruptex" phenotype. Capable of rescuing the lethality conferred by all negatively complementing Ax heteroallelic combinations, individuals display an Ax, not wild type, phenotype.

(Xu et al., 1990)

Wing phenotype: wing vein 2 and the distal part of wing vein 5 are irregularly broadened. Other veins often form knots. All veins have pronounced deltas at the wing margin. Rough eye phenotype. Number of bristles in the thoracic segments is frequently increased.

(Vassin et al., 1985)

Homozygous clones in the eye and the cuticle are cell lethal.

(Dietrich and Campos-Ortega, 1984)

Double mutants Dl^9P and dl² had neuralization of additional ectodermal cells in the thoracic and abdominal segments. Double mutants Dl^9P and Tl had neuralization of the entire ectoderm, a huge CNS and no epidermis as it had been substituted for by neural tissue.

(Campos-Ortega, 1983)

Extreme embryonic neurogenic phenotype.

(Lehmann et al., 1983)

extreme allele

External Data

Linkouts

Interactions

Show genetic interaction network for Enhancers & Suppressors

Phenotypic Class

Enhanced by

Statement

Reference

Dl^9P has visible | dominant phenotype, enhanceable by rumi⁴⁴/rumi⁴⁴

(Acar et al., 2008)

Dl^9P has visible | dominant phenotype, enhanceable by brm[+]/brm²

(Armstrong et al., 2005)

Dl^9P has visible phenotype, enhanceable by neur^UAS.cLa/Scer\GAL4^VMQ

(Lai and Rubin, 2001)

NOT Enhanced by

Statement

Reference

Dl^9P has visible phenotype, non-enhanceable by Scer\GAL4^VMQ/neur^αRING.UAS

(Lai and Rubin, 2001)

Suppressed by

Statement

Reference

Dl^9P has FlyBase_internaltransgene_features:cell lethal:cell lethal | somatic clone phenotype, suppressible by H²

(Dietrich and Campos-Ortega, 1984)

Enhancer of

Statement

Reference

Dl^9P/Dl[+] is an enhancer of visible phenotype of Scer\GAL4^A9, uif^asterisk.UAS

(Xie et al., 2012)

Dl^9P/Dl[+] is an enhancer of visible phenotype of Rala^EE1

(Cho and Fischer, 2011)

Dl^9P/Dl[+] is an enhancer of visible phenotype of rumi⁴⁴

(Acar et al., 2008)

Dl^9P/Dl[+] is an enhancer of visible phenotype of Scer\GAL4^lz-gal4, sens^UAS.cNa

(Pepple et al., 2007)

Dl^9P/Dl[+] is an enhancer of visible phenotype of Scer\GAL4^Tub.PU, csw^N308D.UASp

(Oishi et al., 2006)

Dl^9P/Dl[+] is an enhancer of visible phenotype of Scer\GAL4^ey.PH, brm^{K804R.UAS.Tag:HA}

(Armstrong et al., 2005)

Dl^9P/Dl[+] is an enhancer of visible | dominant phenotype of brm²

(Armstrong et al., 2005)

Dl^9P/Dl[+] is an enhancer of visible | heat sensitive phenotype of peb¹

(Wilk et al., 2004)

Dl^9P is an enhancer of visible phenotype of Scer\GAL4^sca-109-68, ato^UAS.cJa

(White and Jarman, 2000)

Suppressor of

Statement

Reference

Dl^9P is a suppressor of visible phenotype of upd1^GMR.PB

(Mukherjee et al., 2006)

Dl^9P is a suppressor of visible phenotype of Ret^MEN2B.GMR

(Read et al., 2005)

Dl^9P is a suppressor of visible phenotype of Ret^MEN2A.GMR

(Read et al., 2005)

Dl^9P is a suppressor of lethal phenotype of N^Ax-9/N^Ax-E2

(Xu et al., 1990)

Phenotype Manifest In

Enhanced by

Statement

Reference

Dl^9P has wing vein phenotype, enhanceable by rumi⁴⁴/rumi⁴⁴

(Acar et al., 2008)

Dl^9P has macrochaeta phenotype, enhanceable by brm[+]/brm²

(Armstrong et al., 2005)

Dl^9P has wing vein phenotype, enhanceable by Snr1^R3/Snr1^E1

(Marenda et al., 2004)

Dl^9P has wing vein phenotype, enhanceable by Snr1^E1

(Marenda et al., 2004)

Dl^9P has wing vein | ectopic phenotype, enhanceable by ebi^E4

(Tsuda et al., 2002)

Dl^9P has wing vein phenotype, enhanceable by neur^UAS.cLa/Scer\GAL4^VMQ

(Lai and Rubin, 2001)

Dl^9P has phenotype, enhanceable by Su(H)²

(Fortini and Artavanis-Tsakonas, 1994)

Dl^9P has wing phenotype, enhanceable by E(Dl)F14[+]/E(Dl)F14²

(Klein and Campos-Ortega, 1992)

Dl^9P has wing phenotype, enhanceable by E(Dl)F14[+]/E(Dl)F14³

(Klein and Campos-Ortega, 1992)

Dl^9P has wing phenotype, enhanceable by E(Dl)F14⁴/E(Dl)F14[+]

(Klein and Campos-Ortega, 1992)

Dl^9P has wing phenotype, enhanceable by E(Dl)F14[+]/E(Dl)F14^KP77

(Klein and Campos-Ortega, 1992)

Dl^9P has wing phenotype, enhanceable by E(Dl)F14^P/E(Dl)F14[+]

(Klein and Campos-Ortega, 1992)

Dl^9P has wing phenotype, enhanceable by E(Dl)F167[+]/E(Dl)F167¹

(Klein and Campos-Ortega, 1992)

Dl^9P has wing phenotype, enhanceable by E(Dl)F25[+]/E(Dl)F25¹

(Klein and Campos-Ortega, 1992)

Dl^9P has wing phenotype, enhanceable by E(Dl)F42[+]/E(Dl)F42¹

(Klein and Campos-Ortega, 1992)

Dl^9P has wing phenotype, enhanceable by RpL19[+]/RpL19^P

(Klein and Campos-Ortega, 1992)

Dl^9P has wing phenotype, enhanceable by E(Dl)KP254^P/E(Dl)KP254[+]

(Klein and Campos-Ortega, 1992)

Dl^9P has wing phenotype, enhanceable by E(Dl)KP420^P

(Klein and Campos-Ortega, 1992)

Dl^9P has wing phenotype, enhanceable by Ni[+]/Ni-K^D115

(Klein and Campos-Ortega, 1992)

Dl^9P has wing phenotype, enhanceable by Ni-K^F200/Ni[+]

(Klein and Campos-Ortega, 1992)

Dl^9P has wing phenotype, enhanceable by Px[+]/Plexate¹⁰

(Klein and Campos-Ortega, 1992)

Dl^9P has wing phenotype, enhanceable by Px[+]/Plexate¹¹

(Klein and Campos-Ortega, 1992)

Dl^9P has wing phenotype, enhanceable by Px[+]/Plexate⁹

(Klein and Campos-Ortega, 1992)

Dl^9P has wing phenotype, enhanceable by Plexate^D10/Px[+]

(Klein and Campos-Ortega, 1992)

Dl^9P has wing phenotype, enhanceable by Px[+]/Plexate^D123

(Klein and Campos-Ortega, 1992)

Dl^9P has wing phenotype, enhanceable by Px[+]/Plexate^F5

(Klein and Campos-Ortega, 1992)

Dl^9P has wing phenotype, enhanceable by Px[+]/Plexate^F7

(Klein and Campos-Ortega, 1992)

Dl^9P has wing phenotype, enhanceable by E(Dl)9P39.3¹/E(Dl)9P39.3[+]

(Klein and Campos-Ortega, 1992)

Dl^9P has wing phenotype, enhanceable by bs^F31/bs[+]

(Klein and Campos-Ortega, 1992)

Dl^9P has wing phenotype, enhanceable by net[+]/net^9P39.6

(Klein and Campos-Ortega, 1992)

Dl^9P has wing phenotype, enhanceable by net^D90/net[+]

(Klein and Campos-Ortega, 1992)

Dl^9P has wing phenotype, enhanceable by net[+]/net^F170

(Klein and Campos-Ortega, 1992)

Dl^9P has wing phenotype, enhanceable by net^KP92/net[+]

(Klein and Campos-Ortega, 1992)

Dl^9P has wing phenotype, enhanceable by px[+]/px¹⁹

(Klein and Campos-Ortega, 1992)

Dl^9P has wing phenotype, enhanceable by E(Dl)9P39.8[+]/E(Dl)9P39.8¹

(Klein and Campos-Ortega, 1992)

Dl^9P has wing phenotype, enhanceable by E(Dl)D165¹/E(Dl)D165[+]

(Klein and Campos-Ortega, 1992)

Dl^9P has wing phenotype, enhanceable by cic[+]/cic^D49

(Klein and Campos-Ortega, 1992)

Dl^9P has wing phenotype, enhanceable by E(Dl)D72¹/E(Dl)D72[+]

(Klein and Campos-Ortega, 1992)

Dl^9P has wing phenotype, enhanceable by E(Dl)F10¹/E(Dl)F10[+]

(Klein and Campos-Ortega, 1992)

Dl^9P has wing phenotype, enhanceable by E(Dl)F14¹/E(Dl)F14[+]

(Klein and Campos-Ortega, 1992)

Dl^9P has wing phenotype, enhanceable by RpS3²

(Dietrich and Campos-Ortega, 1984)

NOT Enhanced by

Statement

Reference

Dl^9P has wing vein phenotype, non-enhanceable by Scer\GAL4^VMQ/neur^αRING.UAS

(Lai and Rubin, 2001)

Suppressed by

Statement

Reference

Dl^9P has wing vein phenotype, suppressible | partially by Xxylt^Δcds/Xxylt^Δcds

(Pandey et al., 2018)

Dl^9P has wing phenotype, suppressible by S[+]/S⁵⁵

(Klein and Campos-Ortega, 1992)

Dl^9P has wing phenotype, suppressible by S[+]/S⁵⁶

(Klein and Campos-Ortega, 1992)

Dl^9P has wing phenotype, suppressible by S[+]/S⁶⁵

(Klein and Campos-Ortega, 1992)

Dl^9P has wing phenotype, suppressible by S⁶⁶/S[+]

(Klein and Campos-Ortega, 1992)

Dl^9P has wing phenotype, suppressible by S[+]/S⁶⁷

(Klein and Campos-Ortega, 1992)

Dl^9P has wing phenotype, suppressible by S^KP347/S[+]

(Klein and Campos-Ortega, 1992)

Dl^9P has wing phenotype, suppressible by Su(Dl)D107¹/Su(Dl)D107[+]

(Klein and Campos-Ortega, 1992)

Dl^9P has wing phenotype, suppressible by Su(Dl)F116¹/Su(Dl)F116[+]

(Klein and Campos-Ortega, 1992)

Dl^9P has wing phenotype, suppressible by S[+]/S⁵⁷

(Klein and Campos-Ortega, 1992)

Dl^9P has wing phenotype, suppressible by S[+]/S⁵⁸

(Klein and Campos-Ortega, 1992)

Dl^9P has wing phenotype, suppressible by S[+]/S⁵⁹

(Klein and Campos-Ortega, 1992)

Dl^9P has wing phenotype, suppressible by S⁶⁰/S[+]

(Klein and Campos-Ortega, 1992)

Dl^9P has wing phenotype, suppressible by S[+]/S⁶¹

(Klein and Campos-Ortega, 1992)

Dl^9P has wing phenotype, suppressible by S⁶²/S[+]

(Klein and Campos-Ortega, 1992)

Dl^9P has wing phenotype, suppressible by S⁶³/S[+]

(Klein and Campos-Ortega, 1992)

Dl^9P has wing phenotype, suppressible by S[+]/S⁶⁴

(Klein and Campos-Ortega, 1992)

Dl^9P has eye | somatic clone phenotype, suppressible by H²

(Dietrich and Campos-Ortega, 1984)

Dl^9P has presumptive embryonic/larval central nervous system phenotype, suppressible by H²

(Dietrich and Campos-Ortega, 1984)

NOT suppressed by

Statement

Reference

Dl^9P has embryonic epidermis phenotype, non-suppressible by bib^UAS.cDa/Scer\GAL4^h-1J3

(Doherty et al., 1997)

Enhancer of

Statement

Reference

Dl^9P/Dl[+] is an enhancer of wing vein phenotype of Scer\GAL4^A9, uif^asterisk.UAS

(Xie et al., 2012)

Dl^9P/Dl[+] is an enhancer of eye phenotype of Rala^EE1

(Cho and Fischer, 2011)

Dl^9P/Dl[+] is an enhancer of wing vein phenotype of rumi⁴⁴

(Acar et al., 2008)

Dl^9P/Dl[+] is an enhancer of interommatidial bristle | ectopic phenotype of Scer\GAL4^lz-gal4, sens^UAS.cNa

(Pepple et al., 2007)

Dl^9P/Dl[+] is an enhancer of ommatidium phenotype of Scer\GAL4^lz-gal4, sens^UAS.cNa

(Pepple et al., 2007)

Dl^9P/Dl[+] is an enhancer of wing vein | ectopic phenotype of Scer\GAL4^Tub.PU, csw^N308D.UASp

(Oishi et al., 2006)

Dl^9P/Dl[+] is an enhancer of eye phenotype of Scer\GAL4^ey.PH, brm^{K804R.UAS.Tag:HA}

(Armstrong et al., 2005)

Dl^9P/Dl[+] is an enhancer of ommatidium phenotype of Scer\GAL4^ey.PH, brm^{K804R.UAS.Tag:HA}

(Armstrong et al., 2005)

Dl^9P/Dl[+] is an enhancer of macrochaeta phenotype of brm²

(Armstrong et al., 2005)

Dl^9P/Dl[+] is an enhancer of eye | heat sensitive phenotype of peb¹

(Wilk et al., 2004)

Dl^9P is an enhancer of ommatidium phenotype of Gp150^k11120b/Gp150^P8

(Fetchko et al., 2002)

Dl^9P is an enhancer of ommatidium phenotype of Gp150^k11120b

(Fetchko et al., 2002)

Dl^9P/Dl[+] is an enhancer of eye phenotype of ebi^1-334.GMR

(Tsuda et al., 2002)

Dl^9P is an enhancer of eye phenotype of Scer\GAL4^sca-109-68, ato^UAS.cJa

(White and Jarman, 2000)

Dl^9P is an enhancer of ommatidium phenotype of Scer\GAL4^sca-109-68, ato^UAS.cJa

(White and Jarman, 2000)

Dl^9P is an enhancer of phenotype of sca^MSKF

(Hu et al., 1995)

Suppressor of

Statement

Reference

Dl^9P/Dl[+] is a suppressor of posterior crossvein phenotype of Dg^O86

(Christoforou et al., 2008)

Dl^9P is a suppressor of eye phenotype of upd1^GMR.PB

(Mukherjee et al., 2006)

Dl^9P is a suppressor of eye phenotype of Ret^MEN2B.GMR

(Read et al., 2005)

Dl^9P is a suppressor of eye phenotype of Ret^MEN2A.GMR

(Read et al., 2005)

Other

Statement

Reference

Dl^9P/Dl[+], brm² has ommatidium phenotype

(Armstrong et al., 2005)

Dl^9P, brm² has ommatidium phenotype

(Armstrong et al., 2005)

Dl^9P, bru1^QB/bru1^PD has stalk follicle cell phenotype

(Yan and Macdonald, 2004)

Dl^9P, bru1^QB/bru1^PD has egg chamber phenotype

(Yan and Macdonald, 2004)

Dl^9P, frc^NP0297 has anterior scutellar bristle phenotype

(Goto et al., 2001)

Dl^9P, Scer\GAL4^VMQ, neur^UAS.cLa has wing phenotype

(Lai and Rubin, 2001)

Df(1)sc-B57, Dl^9P has interstitial cell precursor phenotype

(Tepass and Hartenstein, 1995)

Df(1)sc-B57, Dl^9P has adult midgut progenitor cell phenotype

(Tepass and Hartenstein, 1995)

Dl^9P/Dl[+], Su(dx)¹, dx^ENU has ocellus phenotype

(Busseau et al., 1994)

Dl^9P/Dl[+], Su(dx)¹, dx^ENU has wing phenotype

(Busseau et al., 1994)

Dl^9P/Dl[+], Su(dx)[+]/Su(dx)¹, dx^ENU has ocellus phenotype

(Busseau et al., 1994)

Dl^9P/Dl[+], Su(dx)[+]/Su(dx)¹, dx^ENU has wing phenotype

(Busseau et al., 1994)

Dl^9P, Su(dx)[+]/Su(dx)¹, dx^ENU has ocellus phenotype

(Busseau et al., 1994)

Dl^9P, Su(dx)[+]/Su(dx)¹, dx^ENU has wing phenotype

(Busseau et al., 1994)

Dl^9P, RpS3² has eye phenotype

(Dietrich and Campos-Ortega, 1984)

Additional Comments

Genetic Interactions

Statement

Reference

The wing vein phenotype resulting from the expression of uif^{asterisk.Scer\UAS} under the control of Scer\GAL4^A9 is enhanced by Dl^9P.

(Xie et al., 2012)

The rough eye phenotype of Rala^EE1/Y adults is dominantly enhanced by Dl^9P.

(Cho and Fischer, 2011)

Flies that are homozygous for rumi⁴⁴ and heterozygous for Dl^9P show a synergistic increase in wing vein expansion.

(Acar et al., 2008)

The "detached" posterior crossvein phenotype seen in Dg^O86 homozygotes is suppressed by Dl^9P/+.

(Christoforou et al., 2008)

The Dl^9P thick vein phenotype is enhanced by Snr1^E1, especially near the distal tips of veins L3, L4 and L5. The Dl^9P thick vein phenotype is enhanced by Snr1^E1/Snr1^R3.

(Marenda et al., 2004)

Ovarioles in aret^QB/aret^QB ; Dl^9P/+ females form a single large egg chambers with many germline cells. The fusome appears as dots. Most egg chambers have more than 16 germline cells in aret^QB/aret^PD ; Dl^9P/+ ovaries. Three classes of abnormal egg chambers are seen, which are present in roughly equal numbers. In the first class, a large number of germline cells of roughly equal size are enveloped by a single epithelium of follicle cells. The remaining two classes arise from partial fusion of separate cysts; no stalk cells can be detected and the follicle cell layers of different egg chambers remain in contact with each other. In the "anterior/posterior fusion" class, a well-defined linear organisation within individual ovarioles is maintained, and adjacent egg chambers are fused with each other at their anterior and posterior boundaries. In the "random fusion" class, egg chambers are positioned irregularly and can be closely apposed to multiple different egg chambers on lateral as well as on anterior and posterior surfaces. In both the partial fusion classes, each egg chamber contains an oocyte. In the anterior/posterior fusion class the oocyte is present at the posterior of the cyst and in the random fusion class the oocyte is either lateral or posterior relative to the overall polarity of the ovariole.

(Yan and Macdonald, 2004)

The addition of Dl^9P/+ to Gp150^P8/Gp150^k11120b animals increases the proportion of mutant ommatidia from 19.2% to 67.6%. The addition of Dl^9P/+ to Gp150^k11120b/+ animals increases the proportion of mutant ommatidia from 0.19% to 2.7%.

(Fetchko et al., 2002)

the addition of one copy of ebi^E4 enhances the dominant wing vein phenotype seen in Dl^9P/+ animals. The combination of ebi^1-334.GMR and Dl^9P/+ leads to flies with a significantly rough eye phenotype.

(Tsuda et al., 2002)

frc^NP0297 in double heterozygous combination with Dl^9P results in duplications of the anterior scutellar bristles.

(Goto et al., 2001)

The combination of Dl^9P and neur^Scer\UAS.cLa (when driven by Scer\GAL4^VMQ) produces a blistered wing phenotype.

(Lai and Rubin, 2001)

The combination of Dl^9P and neur^Scer\UAS.cLa ( when driven by Scer\GAL4^VMQ) produces a blistered wing phenotype.

(Lai and Rubin, 2001)

Enhances the rough eye phenotype caused by expression of ato^Scer\UAS.cJa under the control of Scer\GAL4^sca-109-68.

(White and Jarman, 2000)

The cuticle defect of homozygous embryos is not rescued by bib^Scer\UAS.cDa expressed under the control of Scer\GAL4^h-1J3.

(Doherty et al., 1997)

Double mutants with Df(1)sc-B57 have normal midgut epithelium, but the large basophilic midgut cells are missing and the number of adult midgut precursor cells are reduced.

(Tepass and Hartenstein, 1995)

Flies that are dx^ENU/Y; Su(dx)¹/+; Dl^9P/+ are viable but display clear dx phenotypes in the ocelli and wings.

(Busseau et al., 1994)

Delta phenotype dominantly enhanced by Su(H)².

(Fortini and Artavanis-Tsakonas, 1994)

Dl^9P flies carrying RpS3² in trans have an enhanced Dl wing phenotype and extremely rough eyes. Few flies of this genotype eclose. H² reduces the central nervous system hypertrophy seen in Dl^9P homozygous embryos. Double homozygous Dl^9P H² clones in the eye are viable, and are sometimes indistinguishable from wild-type.

(Dietrich and Campos-Ortega, 1984)

Xenogenetic Interactions

Statement

Reference

Complementation and Rescue Data

Comments

Images (0)

Mutant

Wild-type

Stocks (2)

Bloomington

4220

ru¹ h¹ Diap1¹ st¹ cu¹ sr¹ Dl^9P e^s ca¹/TM3, Sb¹ Ser¹

Kyoto

107640

ru¹ h¹ Diap1¹ st¹ cu¹ sr¹ Dl^9P e^s ca¹/TM3, Sb¹

Notes on Origin

Discoverer

Nusslein-Volhard.

Comments

Autosomal repressor: suppress phenotypes of all viable "abruptex" alleles but not the lethality of the lethal "abruptex" alleles.

(Xu et al., 1990)

Strong Dl allele.

(Heitzler and Simpson, 1991)

dominantly suppressed by alleles of H and S and two Su(Dl) loci, and dominantly enhanced by alleles of Px, px, Ni, bs, net and 13 E(Dl) loci.

(Klein and Campos-Ortega, 1992)

The genotypes Dl^RF/Dl^RF, Dl^RF/Dl^6B, and Dl^RF/Dl^9P show a hypomorphic series, with Dl^RF/Dl^9P showing the least function at any given restrictive temperature.

(Parks and Muskavitch, 1993)

External Crossreferences and Linkouts ( 0 )

Synonyms and Secondary IDs (7)

Reported As

Symbol Synonym

9P39

(Mukherjee et al., 2006)

Delta9p

(Grigorian et al., 2011)

Delta^9P

(Zhu et al., 2017)

Dl^9P39

(Lai and Rubin, 2001, Ramain et al., 2001, Renaud and Simpson, 2001, Barolo et al., 2000, Lecourtois and Schweisguth, 1998, Doherty et al., 1997, Sun and Artavanis-Tsakonas, 1997, Hartenstein et al., 1996, Leviten and Posakony, 1996, Baker and Zitron, 1995, Hu et al., 1995, Sturtevant and Bier, 1995, Tepass and Hartenstein, 1995, Busseau et al., 1994, Fortini and Artavanis-Tsakonas, 1994, Rhyu et al., 1994, Bender et al., 1993, Bodmer et al., 1993, Coyle-Thompson and Banerjee, 1993, de Celis et al., 1993, Heitzler and Simpson, 1993, Parks and Muskavitch, 1993, Simpson et al., 1993, Baker and Rubin, 1992, Simpson et al., 1992, Goriely et al., 1991, Heitzler and Simpson, 1991, Rao et al., 1991, Brand and Campos-Ortega, 1990, Xu and Artavanis-Tsakonas, 1990, Xu et al., 1990, de la Concha et al., 1988, Vassin and Campos-Ortega, 1987, Vassin et al., 1985, Dietrich and Campos-Ortega, 1984, Campos-Ortega, 1983, Lehmann et al., 1983)

Dl^9P

(Pandey et al., 2018, LeBon et al., 2014, Xie et al., 2012, Cho and Fischer, 2011, Park et al., 2011, Udolph et al., 2009, Acar et al., 2008, Christoforou et al., 2008, Pepple et al., 2007, Oishi et al., 2006, Brodu et al., 2004)

Dl^9p39

(Lai et al., 2001, Zeng et al., 1998)

Dl^9p

(Liu et al., 2006)

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