Extra EPCs (eve-positive heart associated or pericardial cells) and DA1 founders (and subsequently muscles) are seen in most hemisegments of mutant embryos.
Heterozygotes show wing vein thickenings of L2 and terminal deltas.
The number of ftz expressing MP2 neurons increases compared to wild-type (About 15 on each side of the midline, as compared to 2 in wild-type) in homozygous embryos derived from homozygous female germ-line clones (lacking both maternal and zygotic function). In homozygous embryos derived from homozygous female germ-line clones (lacking both maternal and zygotic function), RP2sib neurons become eve expressing RP2 neurons. Occasionally a third RP2-like cell is observed. Usib neurons also appear to become U neurons.
The number of cells in the nau-expressing muscle precursor clusters is increased compared to wild-type in homozygous embryos and in homozygous embryos derived from homozygous female germline clones (lacking both maternal and zygotic function).
Homozygotes exhibit ommatidia containing an increased number of rhabdomere-bearing cells.
Extreme neurogenic phenotype.
DlX shows severe neural hypertrophy, a 6--9 fold increase in nau expressing cells per cluster relative to wild type. Clusters of βTub85D producing cells also enlarge and merge together but they cannot assume correct morphology as they don't fuse properly, they are also displaced. In addition Mhc protein is highly expressed and the Mhc expressing cells are arranged in irregular clusters.