Truncation mutant lacking most of dpp 5' flanking sequences.
Breakpoint allele.
wing vein L2 | distal (with dpps11)
wing vein L4 | distal (with dpps11)
wing vein L5 | distal (with dpps11)
Late embryos fail to evaginate gastric caeca and fail to form secondary midgut constrictions, and the primary midgut constriction is posteriorly displaced.
dpps4 homozygotes lack the second midgut constriction.
dpps4/dppd6, gbb1 has partially lethal phenotype
dpps4/dppd6 has anterior crossvein phenotype, suppressible by gbb1
dpps4/dpp[+] is a non-enhancer of crossvein | somatic clone phenotype of gbb1
dpps4/dpp[+] is a non-enhancer of wing vein L5 | somatic clone phenotype of gbb1
dpps4, wgl-17 has embryonic midgut constriction 2 phenotype
dpps4, wgl-17 has embryonic midgut constriction 3 phenotype
The dpp shortvein phenotype is strongly enhanced, and the tkv phenotype is suppressed, in dpps4 tkv1/dpps8 tkv8 double mutant flies. The rhohs.PSt and Ras64BAct5C.PC ectopic vein phenotypes are suppressed in combination with dpps4/dpps8. dpps4/dpps8 NAx-1 double mutant flies show pronounced wing vein loss. The dpp loss of vein phenotype is epistatic in dpps4/dpps8 Nnd-3 double mutants, and the vein thickening characteristic of Nnd-3 is eliminated.
dpps4/dpps12 is rescued by dppdpp.674
dpps4/dpps22 is partially rescued by dppUAS.cSa
The wings of dppScer\UAS.cSa; Scer\GAL4dpp.3KK flies are covered in ectopic vein tissue. This phenotype is rescued by dpps22/dpps4. The resulting flies have a milder shortvein phenotype than dpps22/dpps4 flies.
dppdpp.674 completely rescues the second midgut constriction phenotype of dpps4/dpps12 mutant embryos.
Belongs to the shv-lnc class alleles. Fails to fully complement disk region mutants.
Allele class: shv-lnc
The alleles that were "shvS2" and "shvS4" proved to be molecularly absolutely identical.
The dpp gene product is expressed in the ectoderm but not in the visceral mesoderm.
Used to examine regulation of dpp-Ecol\lacZ promoter fusions, whose expression depends on endogenous dpp function demonstrating that positive autoregulation maintains dpp expression in the visceral mesoderm.