FB2025_01 , released February 20, 2025
Allele: Dmel\gtYA82
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General Information
Symbol
Dmel\gtYA82
Species
D. melanogaster
Name
FlyBase ID
FBal0005255
Feature type
allele
Associated gene
Dmel\gt
Associated Insertion(s)
Carried in Construct
Also Known As
gtYA
Key Links
Allele class

amorphic allele - genetic evidence

loss of function allele

Mutagen
Nature of the Allele
Allele class

amorphic allele - genetic evidence

(Petschek and Mahowald, 1990)

loss of function allele

(Eldon and Pirrotta, 1991)
Mutagen
ethyl methanesulfonate
(Kraut and Levine, 1991)
Progenitor genotype
Cytology
Description
Mutations Mapped to the Genome
Curation Data
Type
Location
Additional Notes
References
Variant Molecular Consequences
Associated Sequence Data
DDBJ / EMBL / GenBank
DNA sequence
Protein sequence
 
UniProtKB/Swiss-Prot
    UniProtKB/TrEMBL
      Expression Data
      Reporter Expression
      Additional Information
      Statement
      Reference
       
      Marker for
      Reflects expression of
      Reporter construct used in assay
      Human Disease Associations
      Disease Ontology (DO) Annotations
      Models Based on Experimental Evidence ( 0 )
      Disease
      Evidence
      References
      Modifiers Based on Experimental Evidence ( 0 )
      Disease
      Interaction
      References
      Comments on Models/Modifiers Based on Experimental Evidence ( 0 )
       
      Disease-implicated variant(s)
       
      Phenotypic Data
      Phenotypic Class
      Phenotype Manifest In
      Detailed Description
      Statement
      Reference

      Embryos have a mutant salivary gland phenotype.

      (Lammel and Saumweber, 2000)

      Larvae show a typical "buttonhead" phenotype (the head does not complete involution) and also show loss of denticle bands from segments A5 to A8.

      (Wu et al., 1998)

      Nondefective in gonad assembly.

      (Warrior, 1994)

      No obvious changes in the expression domains of zygotic hb or Kr. The kni posterior domain extends posteriorly.

      (Eldon and Pirrotta, 1991)

      Interacts with RpII140wimp maternal effect.

      (Parkhurst and Ish-Horowicz, 1991)

      Loss of labial and labral structures, fails to complete head involution.

      (Mohler et al., 1989)

      Labial segment and anterior head structures are missing.

      (Jack et al., 1988)

      Abnormal or missing head structures.

      (Riley et al., 1987)

      gtYA82 mutant embryos have defects in abdominal segments A5 to A7, and in the head.

      (Tearle and Nusslein-Volhard, 1987)

      Larval phenotype: labial, anterior thoracic, and A5-A7 segment defects.

      (Carroll and Scott, 1986)
      External Data
      Interactions
      Show genetic interaction network for Enhancers & Suppressors
      Phenotypic Class
      Phenotype Manifest In
      Additional Comments
      Genetic Interactions
      Statement
      Reference
      Xenogenetic Interactions
      Statement
      Reference
      Complementation and Rescue Data
      Partially rescued by

      gtYA82 is partially rescued by gtst2.eve.Tag:HA

      (Wu et al., 1998)
      Comments
      Images (0)
      Mutant
      Wild-type
      Stocks (0)
      Notes on Origin
      Discoverer
      Comments
      Comments

      The expression pattern of a variety of homeotic, pair-rule and gap genes in homozygous tll1 mutant embryos has been analysed.

      (Reinitz and Levine, 1990)
      External Crossreferences and Linkouts ( 0 )
      Synonyms and Secondary IDs (6)
      References (29)