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General Information
Symbol
Dmel\KrIf-1
Species
D. melanogaster
Name
FlyBase ID
FBal0005583
Feature type
allele
Associated gene
Associated Insertion(s)
Carried in Construct
Also Known As
If
Mutagen
Nature of the Allele
Mutations Mapped to the Genome
 
Type
Location
Additional Notes
References
Associated Sequence Data
DNA sequence
Protein sequence
 
 
Progenitor genotype
Cytology
Nature of the lesion
Statement
Reference
Expression Data
Reporter Expression
Additional Information
Statement
Reference
 
Marker for
Reflects expression of
Reporter construct used in assay
Human Disease Associations
Disease Ontology (DO) Annotations
Models Based on Experimental Evidence ( 0 )
Disease
Evidence
References
Modifiers Based on Experimental Evidence ( 0 )
Disease
Interaction
References
Comments on Models/Modifiers Based on Experimental Evidence ( 0 )
 
Phenotypic Data
Phenotypic Class
Phenotype Manifest In
Detailed Description
Statement
Reference
Mutants have an eye outgrowth phenotype. Feeding geldanamycin to homozygous females increases the frequency of the ectopic eye outgrowth phenotype by approximately 10-fold.
KrIf-1/+ flies have small rough eyes.
The size of the eye is reduced and the ommatidia ared fused or absent in the ventral part of the eye and irregularly arranged dorsally.
Heterozygotes have small, narrow eyes that are pointed ventrally. In the ventral portion of the eye the facets are fused or absent and dorsally they are irregularly arranged. Eyes are further reduced in size in homozygotes, forming narrow slits with a glossy surface lacking most ommatidia.
The number of ommatidia is abnormal in heterozygotes.
Displays locomotor activity rhythm with an approximately 24h period.
KrIf-1 males that are also mutant for mle4 show a more severe phenotype than their mle4/+ sibs.
KrIf-1 results in a rough eye phenotype which is more extreme in homozygotes. Eye discs are deeply folded and smaller than wild type. Ommatidia begin to form posterior to the morphogenetic furrow but later stages of ommatidial development are more rarely seen. The adult eye is greatly reduced in size and is made up of pigment cells and lacunae. Photoreceptor rhabdomeres are only rarely found, and have unusual morphology. The eye region sometimes differentiates cuticle.
The mutant phenotype of KrIf-1 is due to a developmental defect in precursor cells. When heterozygous with apx1 the adult eye is half the normal size, the ventral half more prone to irregularities. Eye disc has a fairly large number of clusters with irregular arrangements. When heterozygous with st adult eye is narrow and ventral half is devoid of facets. Rhabdomeres are fused and only 20 ommatidia are present. Eye disc size is reduced with few clusters.
The eye area is reduced to approximately one-half that of wild-type in heterozygotes, it is oblong and generally pointed ventrally. The facets are irregularly distributed, are frequently missing across the middle of the eye, and are sometimes fused or absent in the ventral portion. The eye is a narrow slit in homozygotes and has a smooth glossy surface. Viability and fecundity of both hetero- and homozygotes is good to excellent.
In heterozygote, eye area about one-half normal; narrow and pointed ventrally; facets irregular and often missing across middle of eyes, sometimes fused or absent in ventral portion. In homozygote, eyes are narrow slits with smooth glossy surface. In the eye disk of late third instar larvae, fairly large number of cell clusters in irregular arrangement, especially in ventral half of disk (Renfranz and Benzer, 1989). Viability and fertility good. RK1.
External Data
Interactions
Show genetic interaction network for Enhancers & Suppressors
Phenotypic Class
Enhanced by
Statement
Reference
KrIf-1 has visible phenotype, enhanceable by piwi[+]/piwi1
KrIf-1 has visible phenotype, enhanceable by piwi[+]/piwi2
KrIf-1 has visible phenotype, enhanceable by Hop[+]/Stip1k00616
KrIf-1 has visible | dominant phenotype, enhanceable by Df(2L)E55
KrIf-1 has visible | dominant phenotype, enhanceable by Df(2R)Px2
KrIf-1 has visible | dominant phenotype, enhanceable by Df(2R)bw-S46
KrIf-1 has visible | dominant phenotype, enhanceable by Df(2R)bw5
KrIf-1 has visible | dominant phenotype, enhanceable by Df(2R)cn9
KrIf-1 has visible | dominant phenotype, enhanceable by GstS104227
KrIf-1 has visible | dominant phenotype, enhanceable by In(2LR)Px4
KrIf-1 has visible | dominant phenotype, enhanceable by Df(2R)pk78s
KrIf-1 has visible | dominant phenotype, enhanceable by Kr-h110642
KrIf-1 has visible | dominant phenotype, enhanceable by Mi-21
KrIf-1 has visible | dominant phenotype, enhanceable by Rab608323
KrIf-1 has visible | dominant phenotype, enhanceable by Df(2L)Prl
KrIf-1 has visible | dominant phenotype, enhanceable by S05671
KrIf-1 has visible | dominant phenotype, enhanceable by S07056
KrIf-1 has visible | dominant phenotype, enhanceable by Sema2a03021
KrIf-1 has visible | dominant phenotype, enhanceable by crol10639
KrIf-1 has visible | dominant phenotype, enhanceable by dpp10638
KrIf-1 has visible | dominant phenotype, enhanceable by ex01270
KrIf-1 has visible | dominant phenotype, enhanceable by ex02278
KrIf-1 has visible | dominant phenotype, enhanceable by CCT803996
KrIf-1 has visible | dominant phenotype, enhanceable by chinmo04111
KrIf-1 has visible | dominant phenotype, enhanceable by l(2)0454804548
KrIf-1 has visible | dominant phenotype, enhanceable by Df(2R)PC4
KrIf-1 has visible | dominant phenotype, enhanceable by l(2)0849208492
KrIf-1 has visible | dominant phenotype, enhanceable by osa00090
KrIf-1 has visible | dominant phenotype, enhanceable by styΔ5
KrIf-1 has visible | dominant phenotype, enhanceable by Mi-21/Mi-2[+]
KrIf-1 has visible | dominant phenotype, enhanceable by osa00090/osa[+]
KrIf-1 has visible | dominant phenotype, enhanceable by Fer2LCH[+]/Fer2LCH07016
KrIf-1 has visible | dominant phenotype, enhanceable by Df(3L)brm11
KrIf-1 has visible | dominant phenotype, enhanceable by Df(3R)P115
KrIf-1 has visible | dominant phenotype, enhanceable by Df(3R)kar-D3
KrIf-1 has visible | dominant phenotype, enhanceable by Df(3R)GB104
KrIf-1 has visible | dominant phenotype, enhanceable by Df(3R)p712
KrIf-1 has visible | dominant phenotype, enhanceable by Df(3L)R-G7
KrIf-1 has visible | dominant phenotype, enhanceable by Df(3L)fz-GF3b
KrIf-1 has visible | dominant phenotype, enhanceable by Df(3L)W10
KrIf-1 has visible | dominant phenotype, enhanceable by Df(3L)W4
KrIf-1 has visible | dominant phenotype, enhanceable by Df(3L)kto2
KrIf-1 has visible | dominant phenotype, enhanceable by osa[+]/osa04539
Suppressed by
Statement
Reference
KrIf-1 has visible | dominant phenotype, suppressible by Df(2R)trix
KrIf-1 has visible | dominant phenotype, suppressible by Sc205634
KrIf-1 has visible | dominant phenotype, suppressible by Vha55j2E9
KrIf-1 has visible | dominant phenotype, suppressible by bun05479
KrIf-1 has visible | dominant phenotype, suppressible by bun06475
KrIf-1 has visible | dominant phenotype, suppressible by caps02937
KrIf-1 has visible | dominant phenotype, suppressible by emc05592
KrIf-1 has visible | dominant phenotype, suppressible by styL1
KrIf-1 has visible | dominant phenotype, suppressible by bun02687
KrIf-1 has visible | dominant phenotype, suppressible by Eip75B07041
KrIf-1 has visible | dominant phenotype, suppressible by In(2L)05887
KrIf-1 has visible | dominant phenotype, suppressible by Df(3L)Ar12-1
KrIf-1 has visible | dominant phenotype, suppressible by Df(3L)Ar14-8
KrIf-1 has visible | dominant phenotype, suppressible by Df(3L)GN19
KrIf-1 has visible | dominant phenotype, suppressible by Df(3L)M21
KrIf-1 has visible | dominant phenotype, suppressible by Df(3L)HR119
KrIf-1 has visible | dominant phenotype, suppressible by Df(3L)AC1
KrIf-1 has visible | dominant phenotype, suppressible by Df(3L)Ly
KrIf-1 has visible | dominant phenotype, suppressible by Df(3L)H99
KrIf-1 has visible | dominant phenotype, suppressible by Df(3R)M-Kx1
KrIf-1 has visible | dominant phenotype, suppressible by l(3)06240[+]/Psa06240
KrIf-1 has visible | dominant phenotype, suppressible by l(3)05967[+]/l(3)0596705967
KrIf-1 has visible | dominant phenotype, suppressible by l(3)neo3[+]/l(3)neo31
KrIf-1 has visible | dominant phenotype, suppressible by emc[+]/emc05592
KrIf-1 has visible | dominant phenotype, suppressible by Sc2[+]/Sc205634
KrIf-1 has visible | dominant phenotype, suppressible by Sap13006924/l(3)06924[+]
KrIf-1 has visible | dominant phenotype, suppressible by caps02937/caps[+]
KrIf-1 has visible | dominant phenotype, suppressible by Eip75B[+]/Eip75B07041
KrIf-1 has visible | dominant phenotype, suppressible by rept[+]/rept06945
KrIf-1 has visible | dominant phenotype, suppressible by Vha55j2E9/Vha55[+]
KrIf-1 has visible | dominant phenotype, suppressible by l(3)j2C3[+]/l(3)j2C3j2C3
KrIf-1 has visible | dominant phenotype, suppressible by l(3)neo391/l(3)neo39[+]
KrIf-1 has visible | dominant phenotype, suppressible by l(3)neo47[+]/l(3)neo471
Phenotype Manifest In
Enhanced by
Statement
Reference
KrIf-1 has eye phenotype, enhanceable by piwi[+]/piwi1
KrIf-1 has eye phenotype, enhanceable by piwi[+]/piwi2
KrIf-1 has eye phenotype, enhanceable by Hop[+]/Stip1k00616
KrIf-1 has eye phenotype, enhanceable by Df(2R)Px2
KrIf-1 has ommatidium phenotype, enhanceable by Df(2R)Px2
KrIf-1 has eye phenotype, enhanceable by Df(2R)bw-S46
KrIf-1 has ommatidium phenotype, enhanceable by Df(2R)bw-S46
KrIf-1 has eye phenotype, enhanceable by Df(2R)bw5
KrIf-1 has ommatidium phenotype, enhanceable by Df(2R)bw5
KrIf-1 has eye phenotype, enhanceable by Df(2L)E55
KrIf-1 has eye phenotype, enhanceable by Df(2R)cn9
KrIf-1 has ommatidium phenotype, enhanceable by Df(2R)cn9
KrIf-1 has eye phenotype, enhanceable by GstS104227
KrIf-1 has ommatidium phenotype, enhanceable by GstS104227
KrIf-1 has eye phenotype, enhanceable by In(2LR)Px4
KrIf-1 has ommatidium phenotype, enhanceable by In(2LR)Px4
KrIf-1 has eye phenotype, enhanceable by Df(2R)pk78s
KrIf-1 has ommatidium phenotype, enhanceable by Df(2L)E55
KrIf-1 has ommatidium phenotype, enhanceable by Df(2R)pk78s
KrIf-1 has eye phenotype, enhanceable by Kr-h110642
KrIf-1 has ommatidium phenotype, enhanceable by Kr-h110642
KrIf-1 has eye phenotype, enhanceable by Mi-21
KrIf-1 has ommatidium phenotype, enhanceable by Mi-21
KrIf-1 has eye phenotype, enhanceable by Rab608323
KrIf-1 has ommatidium phenotype, enhanceable by Rab608323
KrIf-1 has eye phenotype, enhanceable by S05671
KrIf-1 has ommatidium phenotype, enhanceable by S05671
KrIf-1 has eye phenotype, enhanceable by S07056
KrIf-1 has ommatidium phenotype, enhanceable by S07056
KrIf-1 has eye phenotype, enhanceable by Sema2a03021
KrIf-1 has ommatidium phenotype, enhanceable by Sema2a03021
KrIf-1 has eye phenotype, enhanceable by Df(2L)Prl
KrIf-1 has eye phenotype, enhanceable by crol10639
KrIf-1 has ommatidium phenotype, enhanceable by crol10639
KrIf-1 has eye phenotype, enhanceable by dpp10638
KrIf-1 has ommatidium phenotype, enhanceable by dpp10638
KrIf-1 has eye phenotype, enhanceable by ex01270
KrIf-1 has ommatidium phenotype, enhanceable by ex01270
KrIf-1 has eye phenotype, enhanceable by ex02278
KrIf-1 has ommatidium phenotype, enhanceable by Df(2L)Prl
KrIf-1 has ommatidium phenotype, enhanceable by ex02278
KrIf-1 has eye phenotype, enhanceable by CCT803996
KrIf-1 has ommatidium phenotype, enhanceable by CCT803996
KrIf-1 has eye phenotype, enhanceable by chinmo04111
KrIf-1 has ommatidium phenotype, enhanceable by chinmo04111
KrIf-1 has eye phenotype, enhanceable by l(2)0454804548
KrIf-1 has ommatidium phenotype, enhanceable by l(2)0454804548
KrIf-1 has eye phenotype, enhanceable by l(2)0849208492
KrIf-1 has ommatidium phenotype, enhanceable by l(2)0849208492
KrIf-1 has eye phenotype, enhanceable by osa00090
KrIf-1 has ommatidium phenotype, enhanceable by osa00090
KrIf-1 has eye phenotype, enhanceable by styΔ5
KrIf-1 has ommatidium phenotype, enhanceable by styΔ5
KrIf-1 has eye phenotype, enhanceable by Df(2R)PC4
KrIf-1 has ommatidium phenotype, enhanceable by Df(2R)PC4
KrIf-1 has eye phenotype, enhanceable by osa[+]/osa04539
KrIf-1 has eye phenotype, enhanceable by Mi-21/Mi-2[+]
KrIf-1 has eye phenotype, enhanceable by osa00090/osa[+]
KrIf-1 has eye phenotype, enhanceable by Fer2LCH[+]/Fer2LCH07016
KrIf-1 has eye phenotype, enhanceable by Df(3L)brm11
KrIf-1 has eye phenotype, enhanceable by Df(3R)P115
KrIf-1 has eye phenotype, enhanceable by Df(3R)kar-D3
KrIf-1 has eye phenotype, enhanceable by Df(3R)GB104
KrIf-1 has eye phenotype, enhanceable by Df(3R)p712
KrIf-1 has eye phenotype, enhanceable by Df(3L)R-G7
KrIf-1 has eye phenotype, enhanceable by Df(3L)fz-GF3b
KrIf-1 has eye phenotype, enhanceable by Df(3L)W10
KrIf-1 has eye phenotype, enhanceable by Df(3L)W4
KrIf-1 has eye phenotype, enhanceable by Df(3L)kto2
NOT Enhanced by
Statement
Reference
KrIf-1 has eye phenotype, non-enhanceable by Low1
Suppressed by
Statement
Reference
KrIf-1 has eye phenotype, suppressible by Sc205634
KrIf-1 has ommatidium phenotype, suppressible by Sc205634
KrIf-1 has eye phenotype, suppressible by Vha55j2E9
KrIf-1 has ommatidium phenotype, suppressible by Vha55j2E9
KrIf-1 has eye phenotype, suppressible by bun05479
KrIf-1 has ommatidium phenotype, suppressible by bun05479
KrIf-1 has eye phenotype, suppressible by Df(2R)trix
KrIf-1 has eye phenotype, suppressible by bun06475
KrIf-1 has ommatidium phenotype, suppressible by bun06475
KrIf-1 has eye phenotype, suppressible by caps02937
KrIf-1 has ommatidium phenotype, suppressible by caps02937
KrIf-1 has eye phenotype, suppressible by emc05592
KrIf-1 has ommatidium phenotype, suppressible by emc05592
KrIf-1 has eye phenotype, suppressible by styL1
KrIf-1 has ommatidium phenotype, suppressible by Df(2R)trix
KrIf-1 has ommatidium phenotype, suppressible by styL1
KrIf-1 has eye phenotype, suppressible by bun02687
KrIf-1 has ommatidium phenotype, suppressible by bun02687
KrIf-1 has eye phenotype, suppressible by Eip75B07041
KrIf-1 has ommatidium phenotype, suppressible by Eip75B07041
KrIf-1 has eye phenotype, suppressible by In(2L)05887
KrIf-1 has ommatidium phenotype, suppressible by In(2L)05887
KrIf-1 has eye phenotype, suppressible by Df(3L)Ar12-1
KrIf-1 has eye phenotype, suppressible by Df(3L)Ar14-8
KrIf-1 has eye phenotype, suppressible by Df(3L)GN19
KrIf-1 has eye phenotype, suppressible by Df(3L)M21
KrIf-1 has eye phenotype, suppressible by Df(3L)HR119
KrIf-1 has eye phenotype, suppressible by Df(3L)AC1
KrIf-1 has eye phenotype, suppressible by Df(3L)Ly
KrIf-1 has eye phenotype, suppressible by Df(3L)H99
KrIf-1 has eye phenotype, suppressible by Df(3R)M-Kx1
KrIf-1 has eye phenotype, suppressible by l(3)06240[+]/Psa06240
KrIf-1 has eye phenotype, suppressible by l(3)05967[+]/l(3)0596705967
KrIf-1 has eye phenotype, suppressible by l(3)neo3[+]/l(3)neo31
KrIf-1 has eye phenotype, suppressible by emc[+]/emc05592
KrIf-1 has eye phenotype, suppressible by Sc2[+]/Sc205634
KrIf-1 has eye phenotype, suppressible by Sap13006924/l(3)06924[+]
KrIf-1 has eye phenotype, suppressible by caps02937/caps[+]
KrIf-1 has eye phenotype, suppressible by Eip75B[+]/Eip75B07041
KrIf-1 has eye phenotype, suppressible by rept[+]/rept06945
KrIf-1 has eye phenotype, suppressible by Vha55j2E9/Vha55[+]
KrIf-1 has eye phenotype, suppressible by l(3)j2C3[+]/l(3)j2C3j2C3
KrIf-1 has eye phenotype, suppressible by l(3)neo391/l(3)neo39[+]
KrIf-1 has eye phenotype, suppressible by l(3)neo47[+]/l(3)neo471
KrIf-1 has eye phenotype, suppressible by exba[+]/kra03022
NOT suppressed by
Statement
Reference
KrIf-1 has eye phenotype, non-suppressible by Low1
Enhancer of
Statement
Reference
KrIf-1/Kr[+] is an enhancer of eye phenotype of Scer\GAL4GMR.PF, fruNP0021
Other
Statement
Reference
KrIf-1, kto[+]/kto1 has eye phenotype
KrIf-1, osa2/osa[+] has eye phenotype
KrIf-1, skd2/skd[+] has eye phenotype
KrIf-1, Nsf2urd-2/urd[+] has eye phenotype
KrIf-1, vtd[+]/vtd3 has eye phenotype
KrIf-1, z[+]/zv77h has eye phenotype
Hsp83e4A/Hsp83[+], KrIf-1 has eye phenotype
Hsp83[+]/Hsp83e6A, KrIf-1 has eye phenotype
Hsp83e6D/Hsp83[+], KrIf-1 has eye phenotype
KrIf-1, Trl[+]/TrlR85 has eye phenotype
KrIf-1, brm[+]/brm2 has eye phenotype
KrIf-1, btldev2/btl[+] has eye phenotype
KrIf-1, fs(1)h1/fs(1)h[+] has eye phenotype
Additional Comments
Genetic Interactions
Statement
Reference
piwi1/+ and piwi2/+ enhance the eye outgrowth phenotype phenotype of KrIf-1; approximately 7% of the progeny have an eye outgrowth phenotype in a cross of piwi1/+ or piwi2/+ females to KrIf-1/+ males. However, the reciprocal cross of KrIf-1/+ females to piwi1/+ or piwi2/+ males does not result in progeny with an eye outgrowth phenotype, indicating a maternal effect. In addition, the expression of the eye outgrowth phenotype depends on the presence of the piwi2 mutation in the progeny, indicating a zygotic effect. The ability of geldanamycin to increase the frequency of the ectopic eye outgrowth phenotype in KrIf-1 homozygous females is reduced if the females are also carrying piwi5'T:Hsap\MYC. Maternal Hopk00616 is a dominant enhancer of the eye outgrowth phenotype phenotype of KrIf-1.
A KrIf-1 background strongly enhances the rough eye phenotype found upon expression of fruNP0021 under the control of Scer\GAL4GMR.PF.
15+/-4% of KrIf-1/+ flies born to Hsp83e1D/+ mothers have outgrowths with ectopic vibrissae protruding from the ventral region of the eye, compared to less than 0.1% of KrIf-1/+ flies born to isogenised wild-type mothers. 11+/-3% of KrIf-1/+ flies born to Hsp83e3A/+ mothers have outgrowths with ectopic vibrissae protruding from the ventral region of the eye, compared to less than 0.1% of KrIf-1/+ flies born to isogenised wild-type mothers. 6+/-2% of KrIf-1/+ flies born to Hsp83e4A/+ mothers have outgrowths with ectopic vibrissae protruding from the ventral region of the eye, compared to less than 0.1% of KrIf-1/+ flies born to isogenised wild-type mothers. 6+/-2% of KrIf-1/+ flies born to Hsp83e6A/+ mothers have outgrowths with ectopic vibrissae protruding from the ventral region of the eye, compared to less than 0.1% of KrIf-1/+ flies born to isogenised wild-type mothers. 13+/-4% of KrIf-1/+ flies born to Hsp83e6D/+ mothers have outgrowths with ectopic vibrissae protruding from the ventral region of the eye, compared to less than 0.1% of KrIf-1/+ flies born to isogenised wild-type mothers. 5+/-1% of KrIf-1/+ flies born to brm2/+ mothers have outgrowths with ectopic vibrissae protruding from the ventral region of the eye, compared to less than 0.1% of KrIf-1/+ flies born to isogenised wild-type mothers. 10+/-2% of KrIf-1/+ flies born to fs(1)h1/+ mothers have outgrowths with ectopic vibrissae protruding from the ventral region of the eye, compared to less than 0.1% of KrIf-1/+ flies born to isogenised wild-type mothers. 15+/-4% of KrIf-1/+ flies born to kto1/+ mothers have outgrowths with ectopic vibrissae protruding from the ventral region of the eye, compared to less than 0.1% of KrIf-1/+ flies born to isogenised wild-type mothers. 10+/-2% of KrIf-1/+ flies born to osa2/+ mothers have outgrowths with ectopic vibrissae protruding from the ventral region of the eye, compared to less than 0.1% of KrIf-1/+ flies born to isogenised wild-type mothers. 2+/-1% of KrIf-1/+ flies born to skd2/+ mothers have outgrowths with ectopic vibrissae protruding from the ventral region of the eye, compared to less than 0.1% of KrIf-1/+ flies born to isogenised wild-type mothers. 5+/-2% of KrIf-1/+ flies born to urd2/+ mothers have outgrowths with ectopic vibrissae protruding from the ventral region of the eye, compared to less than 0.1% of KrIf-1/+ flies born to isogenised wild-type mothers. 55+/-8% of KrIf-1/+ flies born to vtd3/+ mothers have outgrowths with ectopic vibrissae protruding from the ventral region of the eye, compared to less than 0.1% of KrIf-1/+ flies born to isogenised wild-type mothers. 5+/-2% of KrIf-1/+ flies born to zv77h/+ mothers have outgrowths with ectopic vibrissae protruding from the ventral region of the eye, compared to less than 0.1% of KrIf-1/+ flies born to isogenised wild-type mothers. 10+/-3% of KrIf-1/+ flies born to btldev2/+ mothers have outgrowths with ectopic vibrissae protruding from the ventral region of the eye, compared to less than 0.1% of KrIf-1/+ flies born to isogenised wild-type mothers. 10+/-2% of KrIf-1/+ flies born to TrlR85/+ mothers have outgrowths with ectopic vibrissae protruding from the ventral region of the eye, compared to less than 0.1% of KrIf-1/+ flies born to isogenised wild-type mothers.
KrIf-1, ex01270 flies develop smaller eyes and fewer ommatidia than KrIf-1 flies. KrIf-1, S05671 eyes show a severe enhancement of the KrIf-1 phenotype; the eyes only have only remnants of a few ommatidia.
The eye phenotype is unaffected by Low1.
Xenogenetic Interactions
Statement
Reference
Complementation and Rescue Data
Comments
Images (0)
Mutant
Wild-type
Stocks (89)
Notes on Origin
Discoverer
Casey, 16th Dec. 1965.
Casey.
External Crossreferences and Linkouts ( 0 )
Synonyms and Secondary IDs (5)
References (17)