homozygous embryonic heart cardioblasts show a rounded morphology, show significant decreases in migration velocity, as well as in filopodial and lamellopodial extensions and activities, as compared to controls.
stage 13 embryos have gonads with unfused somatic gonadal precursor (SGP) clusters. By stage 15, SGP clusters fuse but gonads then fail to compact properly. Mutants also show germ cell ensheathment defects.
embryos show defective heart development. Heart defects include contralaterally placed cardial cell nuclei, missing cardial cells from rows of migrating cardial cells and a low level of delays in cell migration.