22.5% of heterozygous females have ectopic wing veins.
Heterozygotes have normal wings.
Flies heterozygous for mor1 show suppression of the Scer\GAL4GMR.PS, DrefScer\UAS.cSa severe rough eye phenotype; eyes are modestly rough and fused ommatidia are not apparent.
mor1 clones show a severe reduction in growth phenotype.
Homozygous clones in the haltere can give rise to wing structures; many clones in the posterior haltere differentiate large bristles characteristic of anterior wing margin. Clones induced early in development cause the haltere to be absent, small, or have a homeotic transformation. Clones induced later (48-72 hours after egg laying) show more extreme transformations of haltere to anterior wing structures than early clones. Homozygous clones in the posterior wing cause distortions in wing shape; venation is disrupted and large socketed bristles appear along the posterior wing margin. Homozygous clones in the leg cause the femur and tibia to be short and twisted, and the tarsal segments to be enlarged. Leg clones also contain extra bristles. Clones in the anterior first leg are transformed to anterior second leg or have extra sex comb teeth. Clones in the second leg are twisted, have vesicles of extra tissue, or cause the absence of the entire leg. Tarsal segments are often fused, duplicated or absent. Clones in the third leg often have partial or complete duplications of distal structures, or the entire leg morphology is abnormal. Clones in the first and third legs often cause truncations of the leg. Eggs derived from homozygous germ-line clones fail to hatch and are very abnormal. These abnormalities include collapsed eggs, eggs with partial or no chorions, small or abnormally shaped eggs and fused and thick dorsal appendages.
Mutation changes the level of w expression in ph-plac+3 flies; eye colour is lighter.