Clones induced early in development (24-48 hours after egg laying) are associated with small or absent halteres and the replacement of proximal haltere structures by proximal wing structures. Clones induced later (48-72 hours after egg laying) often lead to the absence or duplication of halteres, in addition to the appearance of wing structures in the haltere. Homozygous clones also cause transformation of the metanotum to mesonotum. Homozygous clones in the posterior wing cause distortions in wing shape; venation is disrupted and large socketed bristles appear along the posterior wing margin. Homozygous clones in the leg cause the femur and tibia to be short and twisted, and the tarsal segments to be enlarged. Leg clones also contain extra bristles. Clones in the anterior first leg are transformed to anterior second leg or have extra sex comb teeth. Clones in the second leg are twisted, have vesicles of extra tissue, or cause the absence of the entire leg. Tarsal segments are often fused, duplicated or absent. Clones in the third leg often have partial or complete duplications of distal structures, or the entire leg morphology is abnormal. Clones in the first and third legs often cause truncations of the leg. Eggs derived from homozygous germ-line clones fail to hatch and are very abnormal. These abnormalities include collapsed eggs, eggs with partial or no chorions, small or abnormally shaped eggs and fused and thick dorsal appendages.
Mutation changes the level of w expression in ph-plac+3 flies; eye colour is lighter.