The pH of the fecal output of mutant females is more similar to that of wild-type virgin males than to that of wild-type virgin females.
Wild-type mated females often excrete fecal deposits which are extremely concentrated, referred to as reproductive oblong deposits (RODs), compared to the fecal deposits of wild-type males and wild-type virgin females. Mated ovoD1 females show reduced ROD production compared to wild-type mated females.
Ovaries from heterozygous ovoD1 females produce only very rarely egg chambers, which then only survive until stages 2-3 of oogenesis.
ovoD1 flies feed more than control flies. This is because they increase the volume of intake per proboscis extension, rather than by feeding at a greater frequency.
ovoD1 mutant germ lines exhibit growth arrest after stage 4 resulting in small stage 6 egg chambers, which later degenerate.
Heterozygous females have an extended life-span compared to controls.
Heterozygous adult females have atrophied ovaries containing some germ cells but lacking vitellogenic egg chambers. Wild-type clones made in the ovaries of a ovoD1 female, are significantly larger than clones of heterozygous mutant cells. This size difference is greater in clones initiated at 48 hours after egg laying (AEL) than 2 hours AEL.
The frequency of gypsy insertion into the ovo locus in a flam permissive background is higher in ovoD1/+ females than in wild-type females. gypsy insertions in ovoD1 females occur during most stages of germ-line development, in contrast to wild-type females where insertions occur only in late stages.
Heterozygous females have small ovaries bearing only early egg chambers.
Oogenesis stops at about stage 4 in heterozygous females.
Females carrying ovoD1 in a wild-type or ovo-/+ background show dominant female sterility. Heterozygous females contain no mature eggs.
In heterozygous females oogenesis is arrested prior to or at stage 4.
Oogenesis in heterozygous females is mainly arrested prior to stage 4.
Heterozygous females do not lay eggs, egg chambers form but degenerate before vitellogenic stages.
The egg chambers of heterozygous females fail to initiate vitellogenesis, and no eggs are laid. The soma is wild-type. Females heterozygous for both ovoD1 and snf1 have germ cells with a male-like morphology.
Mutant gene activity works in opposition to wild type activity. Viable. Male germ line is fertile, female germ line has no vitellogenic egg chambers.
The presence of ovoD1 has no effect on the dl1 phenotype.