Expressing Dsor1^GD9123 under the control of Scer\GAL4^en.PU in a rl^Sem/+ background results in notched wings with a mix of ectopic and lost vein tissue. Wings did not show creases, or the "droopy" phenotype.

(Hall and Verheyen, 2015)

rl^Sem in combination with Ptp61F^4-10/+ or Scer\GAL4^GMR.PU-driven Ptp61F^{dsRNA.Scer\UAS.WIZ} or Ptp61F^GD3571 results in larger than normal eyes.

(Tchankouo-Nguetcheu et al., 2014)

Heterozygous pum⁷ greatly enhances the dominant extra wing vein phenotype of rl^Sem.

(Kim et al., 2012)

rl^Sem/+ does not suppress the lethality of slpr^BS06/Y males.

(Baril et al., 2009)

S6kII^ign-Δ58-1/Y ; rl^Sem/+ double mutant larvae do not have a higher number of boutons at the neuromuscular junction than either single mutant.

(Fischer et al., 2009)

rl^Sem/+ enhances the mutant wing phenotype caused by expression of Mad^Scer\UAS.cNa under the control of Scer\GAL4^vg.int2.1.

(Zeng et al., 2007)

The ectopic wing vein material phenotype of flies expressing rl^Sem is enhanced in Gug⁰³⁹²⁸, Gug³⁵, Gug^1207D6 and Gug¹⁴⁹⁶⁷ heterozygotes. Expression of Gug^{Ubi-p63E.T:Avic\GFP} suppresses the extra wing vein material formation seen in rl^Sem mutants.

(Charroux et al., 2006)

Formation of ectopic vein tissue in rl^Sem/+ animals is enhanced by ash2^I1/+.

(Angulo et al., 2004)

The rl^Sem extra wing vein phenotype is unaffected by the expression of Mkp3^Scer\UAS.cRa under the control of Scer\GAL4^en-GAL4-33. The formation of additional R7 cells seen in rl^Sem flies is almost completely suppressed by the expression of Mkp3^hs.sev.

(Rintelen et al., 2003)

The increase in midline glial cells seen in rl^Sem mutant embryos is slightly enhanced by W^unspecified/W^unspecified (from 6 (n=231) to 6.6 (n=91)). The reduction in midline glial cell numbers seen in comm^Δe39 mutant embryos is suppressed in double mutants with rl^Sem. These rescued midline glial cells are scattered throughout the midline, which lacks axons in these animals.

(Bergmann et al., 2002)

Double mutants of Fas2^e76 rl^Sem do not exhibit a bouton number significantly different from that found in the Fas2^e76 mutant alone.

(Koh et al., 2002)

rl^Sem/+ alone is not sufficient to rescue the reduced eye and head phenotypes of flies that are homozygous for Ras85D^x7b in the eye disc. However, the addition of Ras85D^D38E in combination with rl^Sem results in a substantial rescue of the Ras85D^x7b eye and head size phenotypes; differentiated photoreceptor cells are seen and a substantial fraction of the ommatidia contain the full complement of photoreceptors.

(Halfar et al., 2001)

The addition of one copy of SelD^k11320 to rl^Sem/+ fliers has no effect on the eye phenotype.

(Morey et al., 2001)

Suppresses the wing margin incision and loss of microchaetae phenotypes of lace^k05305/lace² flies.

(Adachi-Yamada et al., 1999)

rl^Sem completely suppresses the PTP-ER^hs.sev phenotype, to the point of producing ectopic R7 cells as seen in mutants in rl^Sem alone.

(Karim and Rubin, 1999)

Enhances the phenotype of E2f^GMR.PD Dp^GMR.PD BacA\p35^GMR.PH flies.

(Staehling-Hampton et al., 1999)

Suppression of W^Ala3.GMR and W^Ala5.GMR is very mild and noticeably less evident than that of W^GMR.PG.

(Bergmann et al., 1998)

At 25[o]C rl^Sem dominantly enhances most of the hyperplastic growth seen in Ras85D^V12.Scer\UAS Scer\GAL4^dpp.blk1 wing discs resulting in a much larger disc with severe disruptions of morphology. Ras85D^{V12.G37.Scer\UAS} Scer\GAL4^dpp.blk1 and rl^Sem act synergistically to produce a small amount of hyperplastic growth in third instar wing discs. Mutants die as pharate adults at 25[o]C and have deletions of tarsal segments and extra wing vein material along L3.

(Karim and Rubin, 1998)

Suppresses the eye reduction phenotype caused by rpr^GMR.PW. Suppresses the eye reduction phenotype caused by W^GMR.PG.

(Kurada and White, 1998)

Appearance of ectopic photoreceptor cells in the adult eye is suppressed by loss of a functional copy of Jra (Jra¹ or Jra^IA109).

(Kockel et al., 1997)

Suppresses the viability of vn^ddd-13 and proliferation defects in vn^L6.

(Schnepp et al., 1996)

TfIIA-S^E73 suppresses the rough eye phenotype of sev^{S11.T:Hsap\MYC}, phl::tor^12D.hs.sev, Ras85D^V12.sev, rl^Sem and svp^2.sev.

(Zeidler et al., 1996)

Enhancer of the dosage-dependent (two or more copies of P{sev-svp1} or P{sev-svp2}) transformation of cone cells into R7 photoreceptors and at a lower frequency R7 cells into outer photoreceptors.

(Begemann et al., 1995)

Eyes show multiple R7 cells, even in a sev^- mutant background. rl^Sem suppresses the reduced viability associated with phl¹², phl¹ and phl¹⁰⁷, and the eye phenotype of phl¹. Homozygous tll¹ embryos from rl^Sem/+ mothers have a tll mutant phenotype. The extra wing vein phenotype of heterozygotes is enhanced in transheterozygotes with dominant Egfr alleles. Flies heterozygous for Egfr^E3 or Egfr^E1 and rl^Sem have smaller eyes than Egfr^E3 or Egfr^E1 heterozygotes alone.

(Brunner et al., 1994)