Wing blade area is reduced by 20% in homozygous sl2 mutant males compared to wild type. This reduction can be explained by an increase in cell density as wing cells are 21% smaller than in controls but cell number is not significantly different.
sl2 mutant flies sometimes show small ectopic wing veins.
Approximately 23% of sl2 mutant ommatidia contain an increased number of R7 photoreceptors compared to wild type.
Germaria mutant for sl2 do not exhibit ectopic spectrosome containing cells.
In the eyes of sl2 homozygous flies, ~22% of ommatidia are misrotated and almost half have between one and three extra R7 cells.
Mutants have 1.253 +/- 0.038 R7 cells per ommatidia.
Light responses in mutant photoreceptors have essentially wild-type properties.
Border cell migration is normal in sl1/sl2 mutants.
Homozygotes show a modest reduction in wing length and a mildly rough eye. In addition, homozygotes show ectopic wing veins, most frequently adjacent to L2 but sometimes connected or adjacent to the posterior crossvein or in the posterior cell. Ommatidial array is disrupted, many ommatidia are abnormally shaped. Eyes show interommatidial bristle defects. Some are missing, some are duplicated and some appear at additional vertices. 51% of ommatidia contain extra inner photoreceptors. A small fraction of ommatidia have one or more extra (2%) or missing (10%) outer photoreceptors. The majority of supernumerary photoreceptors show an R7 identity. Homozygotes contain as many as three more cone cells than wild type, and an extra primary pigment cell is sometimes present.
Similar to sl1 but possibly more extreme. RK2.