/+ animals completely lack compound eyes.
/+ mutants are eyeless or have severely reduced eyes. The more copies of the allele present in the fly, the more severe the eye phenotype is. The eye primordia of soD
mutants displays normal specification and maintenance, but eye discs become arrested at the time of neuronal morphogenesis and relatively late stages of eye development are affected.
Heterozygotes lack compound eyes and the region normally occupied by the eye is replaced by a large nonpigmented and nondifferentiated field of tissue. Heterozygotes show a complete lack of photoreceptor differentiation during larval eye disc development.
Heterozygotes exhibit an extreme eyeless phenotype, this can be rescued by heat induced expression of so
(repetitive heat shocks during third instar).
Heterozygotes display an extreme reduction in eye size.
RK1 Heterozygote has small, glazed eyes.
Heterozygote has very small glazed eyes; more extreme than DrMio
/+/+ has a very clear phenotype, but weaker than that of DrMio
/+ (i.e., about the size of wgGla-1
/+, but not glazed). Heterozygote has excellent viability. homozygous lethal. gain-of-function allele